rdf:type |
|
lifeskim:mentions |
umls-concept:C0009335,
umls-concept:C0017262,
umls-concept:C0021467,
umls-concept:C0021469,
umls-concept:C0024128,
umls-concept:C0025543,
umls-concept:C0035126,
umls-concept:C0185117,
umls-concept:C0312861,
umls-concept:C0332157,
umls-concept:C1456820,
umls-concept:C2911684
|
pubmed:issue |
3
|
pubmed:dateCreated |
2008-3-6
|
pubmed:abstractText |
Immunity to type V collagen [col(V)] contributes to lung transplant rejection. Matrix metalloproteases (MMPs), which are induced by transplant-related ischemia-reperfusion injury (IRI), could expose col(V) and regulate local IRI-induced inflammation.
|
pubmed:grant |
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Feb
|
pubmed:issn |
0041-1337
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:day |
15
|
pubmed:volume |
85
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
417-26
|
pubmed:meshHeading |
pubmed-meshheading:18322435-Animals,
pubmed-meshheading:18322435-Chemotaxis, Leukocyte,
pubmed-meshheading:18322435-Collagen Type V,
pubmed-meshheading:18322435-Down-Regulation,
pubmed-meshheading:18322435-Lung Transplantation,
pubmed-meshheading:18322435-Metalloproteases,
pubmed-meshheading:18322435-Neutrophils,
pubmed-meshheading:18322435-Oncogene Proteins,
pubmed-meshheading:18322435-Rats,
pubmed-meshheading:18322435-Rats, Inbred WKY,
pubmed-meshheading:18322435-Reperfusion Injury,
pubmed-meshheading:18322435-Tumor Necrosis Factor-alpha
|
pubmed:year |
2008
|
pubmed:articleTitle |
Lung transplant ischemia reperfusion injury: metalloprotease inhibition down-regulates exposure of type V collagen, growth-related oncogene-induced neutrophil chemotaxis, and tumor necrosis factor-alpha expression.
|
pubmed:affiliation |
Center for Immunobiology, Indiana University School of Medicine, Indianapolis, IN 46202, USA.
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
|