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rdf:type |
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lifeskim:mentions |
umls-concept:C0014939,
umls-concept:C0035647,
umls-concept:C0035820,
umls-concept:C0086418,
umls-concept:C0109317,
umls-concept:C0752312,
umls-concept:C0812281,
umls-concept:C1150579,
umls-concept:C1333340,
umls-concept:C1366882,
umls-concept:C1370600,
umls-concept:C1415223,
umls-concept:C1514485,
umls-concept:C1705767,
umls-concept:C1705791
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pubmed:issue |
1
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pubmed:dateCreated |
2011-1-6
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pubmed:abstractText |
The decreased incidence of cardiovascular disease in premenopausal women has been attributed, at least partially, to protective effects of estrogens. However, premenopausal women with diabetes mellitus are no longer selectively protected. High-glucose (HG) conditions have previously been shown to abolish the antimitogenic effects of 17?-estradiol (E(2)) in vascular smooth muscle cells (VSMCs).
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/2,3-bis(4-hydroxyphenyl)-propionitri...,
http://linkedlifedata.com/resource/pubmed/chemical/4,4',4''-(4-propyl-((1)H)-pyrazole-1...,
http://linkedlifedata.com/resource/pubmed/chemical/Estrogen Receptor alpha,
http://linkedlifedata.com/resource/pubmed/chemical/Estrogen Receptor beta,
http://linkedlifedata.com/resource/pubmed/chemical/Extracellular Signal-Regulated MAP...,
http://linkedlifedata.com/resource/pubmed/chemical/Glucose,
http://linkedlifedata.com/resource/pubmed/chemical/Nitriles,
http://linkedlifedata.com/resource/pubmed/chemical/Phenols,
http://linkedlifedata.com/resource/pubmed/chemical/Pyrazoles,
http://linkedlifedata.com/resource/pubmed/chemical/Reactive Oxygen Species,
http://linkedlifedata.com/resource/pubmed/chemical/Superoxide Dismutase
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1945-7197
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
96
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
220-8
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pubmed:dateRevised |
2011-2-15
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pubmed:meshHeading |
pubmed-meshheading:20962025-Analysis of Variance,
pubmed-meshheading:20962025-Aorta,
pubmed-meshheading:20962025-Blotting, Western,
pubmed-meshheading:20962025-Cell Proliferation,
pubmed-meshheading:20962025-Cells, Cultured,
pubmed-meshheading:20962025-Estrogen Receptor alpha,
pubmed-meshheading:20962025-Estrogen Receptor beta,
pubmed-meshheading:20962025-Extracellular Signal-Regulated MAP Kinases,
pubmed-meshheading:20962025-Female,
pubmed-meshheading:20962025-Glucose,
pubmed-meshheading:20962025-Humans,
pubmed-meshheading:20962025-Muscle, Smooth, Vascular,
pubmed-meshheading:20962025-Myocytes, Smooth Muscle,
pubmed-meshheading:20962025-Nitriles,
pubmed-meshheading:20962025-Phenols,
pubmed-meshheading:20962025-Phosphorylation,
pubmed-meshheading:20962025-Pyrazoles,
pubmed-meshheading:20962025-Reactive Oxygen Species,
pubmed-meshheading:20962025-Statistics, Nonparametric,
pubmed-meshheading:20962025-Superoxide Dismutase
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pubmed:year |
2011
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pubmed:articleTitle |
Estrogen receptor-? but not -? or GPER inhibits high glucose-induced human VSMC proliferation: potential role of ROS and ERK.
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pubmed:affiliation |
Division of Clinical and Interventional Angiology, University Hospital Bern, Bern, Switzerland. jana.ortmann@insel.ch
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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