| pubmed-article:7541554 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C0600251 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C0814999 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C0007589 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C0521457 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C1511938 | lld:lifeskim |
| pubmed-article:7541554 | lifeskim:mentions | umls-concept:C1514873 | lld:lifeskim |
| pubmed-article:7541554 | pubmed:issue | 5219 | lld:pubmed |
| pubmed-article:7541554 | pubmed:dateCreated | 1995-8-7 | lld:pubmed |
| pubmed-article:7541554 | pubmed:abstractText | CD25 expression occurs early in thymocyte differentiation. The mechanism of induction of CD25 before T cell receptor rearrangement and the importance of this mechanism for T cell development are unknown. In a thymus reconstitution assay, tumor necrosis factor alpha (TNF-alpha) and interleukin-1 alpha (IL-1 alpha), two cytokines produced within the thymic microenvironment, induced CD25 expression on early immature thymocytes. Either TNF-alpha or IL-1 alpha was necessary for further thymocyte maturation and CD4+CD8+ differentiation. In irradiated mice reconstituted with CD117+CD25+ thymocytes, commitment to the T cell lineage was marked by the loss of precursor multipotency. | lld:pubmed |
| pubmed-article:7541554 | pubmed:language | eng | lld:pubmed |
| pubmed-article:7541554 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:7541554 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:7541554 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:7541554 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:7541554 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:7541554 | pubmed:author | pubmed-author:JiangDD | lld:pubmed |
| pubmed-article:7541554 | pubmed:author | pubmed-author:LenardoM JMJ | lld:pubmed |
| pubmed-article:7541554 | pubmed:author | pubmed-author:Zúñiga-Pflück... | lld:pubmed |
| pubmed-article:7541554 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:7541554 | pubmed:day | 30 | lld:pubmed |
| pubmed-article:7541554 | pubmed:volume | 268 | lld:pubmed |
| pubmed-article:7541554 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:7541554 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:7541554 | pubmed:pagination | 1906-9 | lld:pubmed |
| pubmed-article:7541554 | pubmed:dateRevised | 2010-3-8 | lld:pubmed |
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| pubmed-article:7541554 | pubmed:year | 1995 | lld:pubmed |
| pubmed-article:7541554 | pubmed:articleTitle | Requirement for TNF-alpha and IL-1 alpha in fetal thymocyte commitment and differentiation. | lld:pubmed |
| pubmed-article:7541554 | pubmed:affiliation | Laboratory of Immunology, National Institute of Allergy and Infectious Diseases, Bethesda, MD 20892, USA. | lld:pubmed |
| pubmed-article:7541554 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:7541554 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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