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15753898
Source:
http://linkedlifedata.com/resource/pubmed/id/15753898
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89
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Statements in which the resource exists as a subject.
Predicate
Object
rdf:type
pubmed:Citation
lifeskim:mentions
umls-concept:C0015127
,
umls-concept:C0021758
,
umls-concept:C0030685
,
umls-concept:C0069535
,
umls-concept:C0214743
,
umls-concept:C0250604
,
umls-concept:C0391871
,
umls-concept:C0458827
,
umls-concept:C0680255
,
umls-concept:C1267092
,
umls-concept:C1283071
,
umls-concept:C1314792
,
umls-concept:C1963578
pubmed:issue
3
pubmed:dateCreated
2005-3-8
pubmed:abstractText
Eotaxin is implicated in asthmatic eosinophilia. Oncostatin M (OSM) causes eotaxin release from fibroblasts.
pubmed:grant
http://linkedlifedata.com/resource/pubmed/grant/ES00002
,
http://linkedlifedata.com/resource/pubmed/grant/HL04395
,
http://linkedlifedata.com/resource/pubmed/grant/HL33009
,
http://linkedlifedata.com/resource/pubmed/grant/HL67664
,
http://linkedlifedata.com/resource/pubmed/grant/HL68153
pubmed:language
eng
pubmed:journal
http://linkedlifedata.com/resource/pubmed/journal/1275002
pubmed:citationSubset
AIM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/CCL11 protein, human
,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL11
,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokines, CC
,
http://linkedlifedata.com/resource/pubmed/chemical/DNA-Binding Proteins
,
http://linkedlifedata.com/resource/pubmed/chemical/Growth Inhibitors
,
http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-13
,
http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-4
,
http://linkedlifedata.com/resource/pubmed/chemical/Mitogen-Activated Protein Kinases
,
http://linkedlifedata.com/resource/pubmed/chemical/OSM protein, human
,
http://linkedlifedata.com/resource/pubmed/chemical/Oncostatin M
,
http://linkedlifedata.com/resource/pubmed/chemical/Peptides
,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger
,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Interleukin-4
,
http://linkedlifedata.com/resource/pubmed/chemical/STAT3 Transcription Factor
,
http://linkedlifedata.com/resource/pubmed/chemical/STAT3 protein, human
,
http://linkedlifedata.com/resource/pubmed/chemical/Trans-Activators
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
0091-6749
pubmed:author
pubmed-author:FaffeDébora SDS
,
pubmed-author:FlyntLesleyL
,
pubmed-author:ImrichAmyA
,
pubmed-author:JonesMatthew RMR
,
pubmed-author:MellemaMatthewM
,
pubmed-author:MizgerdJoseph PJP
,
pubmed-author:MoorePaul EPE
,
pubmed-author:PanettieriReynold ARAJr
,
pubmed-author:ShoreStephanie ASA
,
pubmed-author:SilvermanEric SES
,
pubmed-author:SubramaniamVenkatV
,
pubmed-author:WhiteheadTimothyT
pubmed:issnType
Print
pubmed:volume
115
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
514-20
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:15753898-Blotting, Western
,
pubmed-meshheading:15753898-Chemokine CCL11
,
pubmed-meshheading:15753898-Chemokines, CC
,
pubmed-meshheading:15753898-DNA-Binding Proteins
,
pubmed-meshheading:15753898-Drug Synergism
,
pubmed-meshheading:15753898-Enzyme-Linked Immunosorbent Assay
,
pubmed-meshheading:15753898-Flow Cytometry
,
pubmed-meshheading:15753898-Growth Inhibitors
,
pubmed-meshheading:15753898-Humans
,
pubmed-meshheading:15753898-Interleukin-13
,
pubmed-meshheading:15753898-Interleukin-4
,
pubmed-meshheading:15753898-Lung
,
pubmed-meshheading:15753898-Mitogen-Activated Protein Kinases
,
pubmed-meshheading:15753898-Muscle, Smooth
,
pubmed-meshheading:15753898-Oncostatin M
,
pubmed-meshheading:15753898-Peptides
,
pubmed-meshheading:15753898-Phosphorylation
,
pubmed-meshheading:15753898-RNA, Messenger
,
pubmed-meshheading:15753898-Receptors, Interleukin-4
,
pubmed-meshheading:15753898-Reverse Transcriptase Polymerase Chain Reaction
,
pubmed-meshheading:15753898-STAT3 Transcription Factor
,
pubmed-meshheading:15753898-Trans-Activators
pubmed:year
2005
pubmed:articleTitle
Oncostatin M causes eotaxin-1 release from airway smooth muscle: synergy with IL-4 and IL-13.
pubmed:affiliation
Physiology Program, Harvard School of Public Health, Boston, Mass 02115, USA.
pubmed:publicationType
Journal Article
,
Research Support, U.S. Gov't, P.H.S.