| pubmed-article:10438795 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0035236 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0025919 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0027950 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0022688 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0876973 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0079189 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C1423842 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C0142326 | lld:lifeskim |
| pubmed-article:10438795 | lifeskim:mentions | umls-concept:C2003874 | lld:lifeskim |
| pubmed-article:10438795 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:10438795 | pubmed:dateCreated | 1999-9-7 | lld:pubmed |
| pubmed-article:10438795 | pubmed:abstractText | BALB/c mice sensitized to vaccinia virus expressed G protein of respiratory syncytial virus (RSV) develop a Th2-type cytokine response and pulmonary eosinophilia when challenged with live RSV. In this study, BALB/c mice were immunized or challenged with an RSV mutant lacking the G and SH proteins or with DNA vaccines coding for RSV G or F protein. F or G protein DNA vaccines were capable of sensitizing for pulmonary eosinophilia. The absence of the G and/or SH protein in the infecting virus resulted in a consistent increase both in pulmonary natural killer cells and in gamma interferon and tumor necrosis factor expression, as well as, with primary infection, a variable increase in neutrophils and CD11b(+) cells. The development of pulmonary eosinophilia in formalin-inactivated RSV-vaccinated mice required the presence of the G and/or SH protein in the challenge virus. These data show that G and/or SH protein has a marked impact on the inflammatory and innate immune response to RSV infection. | lld:pubmed |
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| pubmed-article:10438795 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10438795 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10438795 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10438795 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10438795 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:10438795 | pubmed:issn | 0022-538X | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:MooreDD | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:WinterJJ | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:JonesLL | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:AndersonL JLJ | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:SullenderWW | lld:pubmed |
| pubmed-article:10438795 | pubmed:author | pubmed-author:TrippR ARA | lld:pubmed |
| pubmed-article:10438795 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10438795 | pubmed:volume | 73 | lld:pubmed |
| pubmed-article:10438795 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10438795 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10438795 | pubmed:pagination | 7099-107 | lld:pubmed |
| pubmed-article:10438795 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:10438795 | pubmed:year | 1999 | lld:pubmed |
