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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
1999-4-28
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pubmed:abstractText |
Pemphigus vulgaris (PV) is a potentially life-threatening disease, characterized immunohistologically by IgG deposits and complement activation on the surface of keratinocytes. Complement activation has been implicated in the pathogenesis with C3 deposits in about 90% of patients.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1203-4754
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
3
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
140-4
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pubmed:dateRevised |
2010-3-23
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pubmed:meshHeading |
pubmed-meshheading:10082594-Acantholysis,
pubmed-meshheading:10082594-Cells, Cultured,
pubmed-meshheading:10082594-Complement Activation,
pubmed-meshheading:10082594-Complement C3,
pubmed-meshheading:10082594-Humans,
pubmed-meshheading:10082594-Interleukin-1,
pubmed-meshheading:10082594-Keratinocytes,
pubmed-meshheading:10082594-Pemphigus,
pubmed-meshheading:10082594-RNA, Messenger,
pubmed-meshheading:10082594-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:10082594-Time Factors,
pubmed-meshheading:10082594-Tumor Necrosis Factor-alpha
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pubmed:year |
1999
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pubmed:articleTitle |
In vitro C3 mRNA expression in Pemphigus vulgaris: complement activation is increased by IL-1alpha and TNF-alpha.
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pubmed:affiliation |
Department of Dermatology, University "G.D'Annunzio," Chieti, Italy.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
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