rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
3
|
pubmed:dateCreated |
1994-7-15
|
pubmed:abstractText |
The capacity of human T cell subsets, CD4+ or CD8+ T cells, to produce tumour necrosis factors (TNF-alpha and TNF-beta) upon stimulation with toxic shock syndrome toxin-1 (TSST-1) and the requirement for MHC class II molecules on accessory cells (AC) in the response were investigated. The capacity of CD4+ T cells was much higher than that of CD8+ T cells in TSST-1-induced production of TNF-alpha and TNF-beta. The expression of MHC class II molecules on AC was required in the response.
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pubmed:commentsCorrections |
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1353448,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1356634,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1452333,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1594585,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1612731,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1730929,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-1972165,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2123824,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2185544,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2404064,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2583222,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2784735,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-2995927,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-3127526,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-3312414,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-3498581,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-3981783,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-6112412,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-6200439,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-6392892,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-6403867,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-6972418,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-8097227,
http://linkedlifedata.com/resource/pubmed/commentcorrection/7911750-8387893
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pubmed:language |
eng
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pubmed:journal |
|
pubmed:citationSubset |
IM
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pubmed:chemical |
|
pubmed:status |
MEDLINE
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pubmed:month |
Jun
|
pubmed:issn |
0009-9104
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pubmed:author |
|
pubmed:issnType |
Print
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pubmed:volume |
96
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
422-6
|
pubmed:dateRevised |
2009-11-18
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pubmed:meshHeading |
pubmed-meshheading:7911750-Animals,
pubmed-meshheading:7911750-Antigen-Presenting Cells,
pubmed-meshheading:7911750-Bacterial Toxins,
pubmed-meshheading:7911750-Base Sequence,
pubmed-meshheading:7911750-CD4-Positive T-Lymphocytes,
pubmed-meshheading:7911750-DNA Primers,
pubmed-meshheading:7911750-Enterotoxins,
pubmed-meshheading:7911750-HLA-DR4 Antigen,
pubmed-meshheading:7911750-Humans,
pubmed-meshheading:7911750-Interleukin-2,
pubmed-meshheading:7911750-L Cells (Cell Line),
pubmed-meshheading:7911750-Lymphocyte Activation,
pubmed-meshheading:7911750-Lymphotoxin-alpha,
pubmed-meshheading:7911750-Mice,
pubmed-meshheading:7911750-Molecular Sequence Data,
pubmed-meshheading:7911750-Staphylococcus aureus,
pubmed-meshheading:7911750-Superantigens,
pubmed-meshheading:7911750-T-Lymphocyte Subsets,
pubmed-meshheading:7911750-T-Lymphocytes,
pubmed-meshheading:7911750-Transfection,
pubmed-meshheading:7911750-Tumor Necrosis Factor-alpha
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pubmed:year |
1994
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pubmed:articleTitle |
Production of tumour necrosis factors by human T cells stimulated by a superantigen, toxic shock syndrome toxin-1.
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pubmed:affiliation |
Department of Microbiology and Immunology, Tokyo Women's Medical College, Japan.
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pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
|