pubmed-article:6112044 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C0439849 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C1512035 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C0038590 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C0206518 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
pubmed-article:6112044 | lifeskim:mentions | umls-concept:C0150312 | lld:lifeskim |
pubmed-article:6112044 | pubmed:issue | 1-2 | lld:pubmed |
pubmed-article:6112044 | pubmed:dateCreated | 1981-7-23 | lld:pubmed |
pubmed-article:6112044 | pubmed:abstractText | Submaximal destruction of nigrostriatal dopaminergic projections resulted in a significant (25%) decrease in specific GABA binding in substantia nigra; under these conditions, striatal tyrosine hydroxylase activity was 15-44% of control. In rats with lesions which caused maximal destruction of nigrostriatal dopamine neurons (striatal tyrosine hydroxylase was less than 15% of control), specific GABA binding in substantia nigra was apparently not different from that obtained in intact controls. Two distinct processes may be occurring in response to the destruction of dopamine neurons: (1) the loss of GABA binding sites physically associated with nigral dopamine neurons; and (2) an increase in nigral GABA receptors associated with non-dopaminergic neurons. The latter process may result from a decrease in nigral GABA transmission secondary to the complete loss of dopaminergic synaptic activity in striatum. | lld:pubmed |
pubmed-article:6112044 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:language | eng | lld:pubmed |
pubmed-article:6112044 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:6112044 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:6112044 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:6112044 | pubmed:month | Apr | lld:pubmed |
pubmed-article:6112044 | pubmed:issn | 0006-8993 | lld:pubmed |
pubmed-article:6112044 | pubmed:author | pubmed-author:BükyPP | lld:pubmed |
pubmed-article:6112044 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:6112044 | pubmed:day | 6 | lld:pubmed |
pubmed-article:6112044 | pubmed:volume | 210 | lld:pubmed |
pubmed-article:6112044 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:6112044 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:6112044 | pubmed:pagination | 401-6 | lld:pubmed |
pubmed-article:6112044 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
pubmed-article:6112044 | pubmed:meshHeading | pubmed-meshheading:6112044-... | lld:pubmed |
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pubmed-article:6112044 | pubmed:meshHeading | pubmed-meshheading:6112044-... | lld:pubmed |
pubmed-article:6112044 | pubmed:year | 1981 | lld:pubmed |
pubmed-article:6112044 | pubmed:articleTitle | Relationship between the presence of dopaminergic neurons and GABA receptors in substantia nigra: effects of lesions. | lld:pubmed |
pubmed-article:6112044 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:6112044 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:6112044 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |