| pubmed-article:19950021 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0015576 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0291573 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0029431 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0033684 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0376515 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:19950021 | lifeskim:mentions | umls-concept:C0445450 | lld:lifeskim |
| pubmed-article:19950021 | pubmed:dateCreated | 2009-12-1 | lld:pubmed |
| pubmed-article:19950021 | pubmed:abstractText | Apopotosis of osteoclasts is regulated by the Bcl-2 family protein Bim. Bim is degraded in the course of osteoclast apoptosis, which is regulated by Caspase-3. Osteoclasts generated from caspase-3 -/- mice exhibited a shorter life span and a higher bone-resorbing activity than those generated from normal littermates. These results suggest the important role of Caspase-3-Bim axis in regulating both apoptosis and activation of osteoclasts. | lld:pubmed |
| pubmed-article:19950021 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19950021 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19950021 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19950021 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19950021 | pubmed:issn | 0065-2598 | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:TanakaSakaeS | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:NakamuraKozoK | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:NakayamaKeiic... | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:TakahashiKats... | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:AmanoHitoshiH | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:AkiyamaToruT | lld:pubmed |
| pubmed-article:19950021 | pubmed:author | pubmed-author:WakeyamaHidet... | lld:pubmed |
| pubmed-article:19950021 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:19950021 | pubmed:volume | 658 | lld:pubmed |
| pubmed-article:19950021 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19950021 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19950021 | pubmed:pagination | 111-6 | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:meshHeading | pubmed-meshheading:19950021... | lld:pubmed |
| pubmed-article:19950021 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:19950021 | pubmed:articleTitle | Regulation of osteoclast apoptosis by Bcl-2 family protein Bim and Caspase-3. | lld:pubmed |
| pubmed-article:19950021 | pubmed:affiliation | Department of Orthopaedic Surgery, Faculty of Medicine, The University of Tokyo, Tokyo, Japan. tanakas-ort@h.u-tokyo.ac.jp | lld:pubmed |
| pubmed-article:19950021 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19950021 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:12125 | entrezgene:pubmed | pubmed-article:19950021 | lld:entrezgene |
| entrez-gene:12367 | entrezgene:pubmed | pubmed-article:19950021 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19950021 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19950021 | lld:entrezgene |
