pubmed-article:17606840 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0376466 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0257694 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0752312 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0907532 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0032214 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0600138 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C1424650 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0205390 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0040661 | lld:lifeskim |
pubmed-article:17606840 | lifeskim:mentions | umls-concept:C0205087 | lld:lifeskim |
pubmed-article:17606840 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:17606840 | pubmed:dateCreated | 2007-7-31 | lld:pubmed |
pubmed-article:17606840 | pubmed:abstractText | The role of endothelial nitric oxide synthase (eNOS) in ischemic preconditioning (PC) and cardioprotection is poorly understood. We addressed this issue using a genetic, rather than pharmacological, approach. | lld:pubmed |
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pubmed-article:17606840 | pubmed:language | eng | lld:pubmed |
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pubmed-article:17606840 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:17606840 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17606840 | pubmed:month | Jul | lld:pubmed |
pubmed-article:17606840 | pubmed:issn | 1524-4539 | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:BolliRobertoR | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:GuoYiruY | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:XuanYu-TingYT | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:ZhuYanqingY | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:WangOu-LiOL | lld:pubmed |
pubmed-article:17606840 | pubmed:author | pubmed-author:RokoshGreggG | lld:pubmed |
pubmed-article:17606840 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:17606840 | pubmed:day | 31 | lld:pubmed |
pubmed-article:17606840 | pubmed:volume | 116 | lld:pubmed |
pubmed-article:17606840 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17606840 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17606840 | pubmed:pagination | 535-44 | lld:pubmed |
pubmed-article:17606840 | pubmed:dateRevised | 2011-11-2 | lld:pubmed |
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pubmed-article:17606840 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17606840 | pubmed:articleTitle | Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 pathway. | lld:pubmed |
pubmed-article:17606840 | pubmed:affiliation | Institute of Molecular Cardiology, University of Louisville, Louisville, KY 40292, USA. ytxuan01@louisville.edu | lld:pubmed |
pubmed-article:17606840 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:17606840 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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