pubmed-article:17306581 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C0032961 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C0032043 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C1333887 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C0242614 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C0015938 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C0021666 | lld:lifeskim |
pubmed-article:17306581 | lifeskim:mentions | umls-concept:C1517945 | lld:lifeskim |
pubmed-article:17306581 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:17306581 | pubmed:dateCreated | 2007-4-10 | lld:pubmed |
pubmed-article:17306581 | pubmed:abstractText | Fetal macrosomia is a common complication of maternal diabetes mellitus and is associated with substantial morbidity, but the precise cellular and molecular mechanisms that induce fetal macrosomia are not well understood. The imprinted genes IGF-II and H19 are crucial for placental development and fetal growth. The term placentas from diabetic pregnancies express more insulin-like growth factor II (IGF-II) than those from normal pregnancies. Deregulation of their imprinting status is observed in the macrosomia-associated syndrome, the Beckwith-Wiedemann syndrome. The aim of this study was to determine whether loss of imprinting hence biallelic expression was also a hallmark of macrosomia in diabetic pregnancies. | lld:pubmed |
pubmed-article:17306581 | pubmed:language | eng | lld:pubmed |
pubmed-article:17306581 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:17306581 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17306581 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17306581 | pubmed:month | Apr | lld:pubmed |
pubmed-article:17306581 | pubmed:issn | 1096-6374 | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:DufourPP | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:FontainePP | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:RousseauxJJ | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:ValatA SAS | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:FajardyII | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:DanzeP MPM | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:VambergueAA | lld:pubmed |
pubmed-article:17306581 | pubmed:author | pubmed-author:VandersippeMM | lld:pubmed |
pubmed-article:17306581 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17306581 | pubmed:volume | 17 | lld:pubmed |
pubmed-article:17306581 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17306581 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17306581 | pubmed:pagination | 130-6 | lld:pubmed |
pubmed-article:17306581 | pubmed:dateRevised | 2011-10-7 | lld:pubmed |
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pubmed-article:17306581 | pubmed:year | 2007 | lld:pubmed |
pubmed-article:17306581 | pubmed:articleTitle | No loss of genomic imprinting of IGF-II and H19 in placentas of diabetic pregnancies with fetal macrosomia. | lld:pubmed |
pubmed-article:17306581 | pubmed:affiliation | Department of Endocrinology and Diabetes, Marc Linquette Hospital, CHRU Lille, France. a-vambergue@chru-lille.fr | lld:pubmed |
pubmed-article:17306581 | pubmed:publicationType | Journal Article | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:17306581 | lld:pubmed |