| pubmed-article:17246216 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17246216 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:17246216 | lifeskim:mentions | umls-concept:C0728940 | lld:lifeskim |
| pubmed-article:17246216 | lifeskim:mentions | umls-concept:C0013138 | lld:lifeskim |
| pubmed-article:17246216 | lifeskim:mentions | umls-concept:C0332183 | lld:lifeskim |
| pubmed-article:17246216 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:17246216 | pubmed:dateCreated | 2010-6-28 | lld:pubmed |
| pubmed-article:17246216 | pubmed:abstractText | RpII215(D) (50) (= D50) is a lethal mutation caused by the insertion of a 1.3-kb P element 5' to sequences encoding the largest (215 kilodaltons) subunit of Drosophila RNA polymerase II. In dysgenic males D50 reverted to nonlethality at frequencies ranging from 2.6 to 6.5%. These reversions resulted from loss of P element sequences. Genetic tests of function and restriction enzyme analysis of revertant DNAs revealed that 35% or more of the reversion events were imprecise excisions. Two meiotic mutations that perturb excision repair and postreplication repair (mei-9(a) and mei-41(D5), respectively) had no influence on reversion frequency but may have increased the proportion of imprecise excisions. We suggest that these excisions are by-products of, rather than intermediates in, the transposition process. | lld:pubmed |
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| pubmed-article:17246216 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17246216 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17246216 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
| pubmed-article:17246216 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:17246216 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:HuangS MSM | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:GreenleafA... | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:GyurkovicsHH | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:VoelkerR ARA | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:SearlesL LLL | lld:pubmed |
| pubmed-article:17246216 | pubmed:author | pubmed-author:WiselyG BGB | lld:pubmed |
| pubmed-article:17246216 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17246216 | pubmed:volume | 107 | lld:pubmed |
| pubmed-article:17246216 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17246216 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17246216 | pubmed:pagination | 279-94 | lld:pubmed |
| pubmed-article:17246216 | pubmed:dateRevised | 2010-9-15 | lld:pubmed |
| pubmed-article:17246216 | pubmed:year | 1984 | lld:pubmed |
| pubmed-article:17246216 | pubmed:articleTitle | Frequent Imprecise Excision among Reversions of a P Element-Caused Lethal Mutation in Drosophila. | lld:pubmed |
| pubmed-article:17246216 | pubmed:affiliation | Laboratory of Genetics, National Institute of Environmental Health Sciences, Research Triangle Park, North Carolina 27709. | lld:pubmed |
| pubmed-article:17246216 | pubmed:publicationType | Journal Article | lld:pubmed |
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