| pubmed-article:16380550 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0018787 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0016059 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0036751 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0596902 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0011155 | lld:lifeskim |
| pubmed-article:16380550 | lifeskim:mentions | umls-concept:C0181586 | lld:lifeskim |
| pubmed-article:16380550 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:16380550 | pubmed:dateCreated | 2006-1-4 | lld:pubmed |
| pubmed-article:16380550 | pubmed:abstractText | Serotonin (5-hydroxytryptamine; 5-HT) overproduction is responsible for cardiac valvular disease in patients with carcinoid tumors. Reduced 5-HT inactivation is one proposed mechanism of the valvulopathy observed in individuals treated with the appetite suppressants fenfluramine and phentermine. One key protein limiting systemic availability of 5-HT is the 5-HT transporter (5-HTT) expressed by platelets and pulmonary vascular cells; 5-HTT is responsible for 5-HT uptake and subsequent inactivation of the amine passing through the lung. Here we investigated whether 5-HTT-deficient (5-HTT-KO) mice developed structural and/or functional cardiac abnormalities and valvulopathy. | lld:pubmed |
| pubmed-article:16380550 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16380550 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16380550 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16380550 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:16380550 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:LechatPP | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:HamonMM | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:KirschMM | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:SeifII | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:PascalOO | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:AdnotSS | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:EddahibiSS | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:Benhaiem-Siga... | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:LanfumeyLL | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:HanounNN | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:Mekontso-Dess... | lld:pubmed |
| pubmed-article:16380550 | pubmed:author | pubmed-author:BrouriFF | lld:pubmed |
| pubmed-article:16380550 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:16380550 | pubmed:day | 3 | lld:pubmed |
| pubmed-article:16380550 | pubmed:volume | 113 | lld:pubmed |
| pubmed-article:16380550 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16380550 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16380550 | pubmed:pagination | 81-9 | lld:pubmed |
| pubmed-article:16380550 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
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| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:meshHeading | pubmed-meshheading:16380550... | lld:pubmed |
| pubmed-article:16380550 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16380550 | pubmed:articleTitle | Deficiency of the 5-hydroxytryptamine transporter gene leads to cardiac fibrosis and valvulopathy in mice. | lld:pubmed |
| pubmed-article:16380550 | pubmed:affiliation | Institut National de la Santé et de la Recherche Médicale (INSERM) Unité 651, Département de Physiologie, Université Paris XII, CHU Henri Mondor, Assistance Publique-Hôpitaux de Paris, Créteil, France. | lld:pubmed |
| pubmed-article:16380550 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16380550 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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