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rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0010592,
umls-concept:C0017262,
umls-concept:C0017337,
umls-concept:C0021469,
umls-concept:C0022658,
umls-concept:C0026336,
umls-concept:C0034693,
umls-concept:C0034721,
umls-concept:C0040691,
umls-concept:C0085542,
umls-concept:C0185117,
umls-concept:C0205191,
umls-concept:C1280500,
umls-concept:C2911684
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|
pubmed:issue |
6
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|
pubmed:dateCreated |
2005-12-19
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|
pubmed:abstractText |
Overexpression of transforming growth factor beta1-inducible gene h3 (betaig-h3) is associated with renal scarring in several models of renal disease. We investigated the inhibitory effect of pravastatin on betaig-h3 expression in a rat model of chronic cyclosporin A (CsA)-induced nephropathy.
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pubmed:language |
eng
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pubmed:journal |
|
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pubmed:citationSubset |
IM
|
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:issn |
0250-8095
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
611-20
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pubmed:dateRevised |
2007-2-14
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pubmed:meshHeading |
pubmed-meshheading:16308546-Animals,
pubmed-meshheading:16308546-Blotting, Northern,
pubmed-meshheading:16308546-Cyclosporine,
pubmed-meshheading:16308546-Disease Models, Animal,
pubmed-meshheading:16308546-Gene Expression Regulation,
pubmed-meshheading:16308546-Immunohistochemistry,
pubmed-meshheading:16308546-Kidney,
pubmed-meshheading:16308546-Male,
pubmed-meshheading:16308546-Nephrosclerosis,
pubmed-meshheading:16308546-Nitric Oxide Synthase Type III,
pubmed-meshheading:16308546-Pravastatin,
pubmed-meshheading:16308546-RNA, Messenger,
pubmed-meshheading:16308546-Rats,
pubmed-meshheading:16308546-Rats, Sprague-Dawley,
pubmed-meshheading:16308546-Transforming Growth Factor beta,
pubmed-meshheading:16308546-Transforming Growth Factor beta1
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pubmed:articleTitle |
Inhibitory effect of pravastatin on transforming growth factor beta1-inducible gene h3 expression in a rat model of chronic cyclosporine nephropathy.
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pubmed:affiliation |
Department of Internal Medicine, Xenotransplantation Center, KangNam St. Mary's Hospital, Seoul, Korea.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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