| pubmed-article:12905472 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0205307 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0033414 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0021665 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0220781 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C1883254 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0205195 | lld:lifeskim |
| pubmed-article:12905472 | lifeskim:mentions | umls-concept:C0331858 | lld:lifeskim |
| pubmed-article:12905472 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:12905472 | pubmed:dateCreated | 2003-8-7 | lld:pubmed |
| pubmed-article:12905472 | pubmed:abstractText | Although growth factor therapy could be an attractive method for stimulating the repair of damaged cartilage matrix, there is evidence that with aging and/or with the development of osteoarthritis (OA), articular chondrocytes may become unresponsive to growth factor stimulation. The aim of the current study was to compare the ability of insulin-like growth factor+(IGF-1) and osteogenic protein+(OP-1), alone and in combination, to stimulate human normal and OA chondrocytes in culture. | lld:pubmed |
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| pubmed-article:12905472 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12905472 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12905472 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12905472 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12905472 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:12905472 | pubmed:issn | 0004-3591 | lld:pubmed |
| pubmed-article:12905472 | pubmed:author | pubmed-author:LoeserRichard... | lld:pubmed |
| pubmed-article:12905472 | pubmed:author | pubmed-author:ChubinskayaSu... | lld:pubmed |
| pubmed-article:12905472 | pubmed:author | pubmed-author:PacioneCarol... | lld:pubmed |
| pubmed-article:12905472 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12905472 | pubmed:volume | 48 | lld:pubmed |
| pubmed-article:12905472 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12905472 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12905472 | pubmed:pagination | 2188-96 | lld:pubmed |
| pubmed-article:12905472 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:12905472 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12905472 | pubmed:articleTitle | The combination of insulin-like growth factor 1 and osteogenic protein 1 promotes increased survival of and matrix synthesis by normal and osteoarthritic human articular chondrocytes. | lld:pubmed |
| pubmed-article:12905472 | pubmed:affiliation | Rheumatology, Rush Medical College of Rush-Presbyterian-St. Luke's Medical Center, 1725 West Harrison, Suite 1017, Chicago, IL 60612, USA. rloeser@rush.edu | lld:pubmed |
| pubmed-article:12905472 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12905472 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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