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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2000-2-3
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pubmed:abstractText |
To verify the hypothesis that in rheumatoid arthritis (RA), tumor necrosis factor alpha (TNFalpha) plays a critical role in regulating leukocyte trafficking and chemokine levels.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Antibodies, Monoclonal,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD22,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, CD3,
http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Differentiation...,
http://linkedlifedata.com/resource/pubmed/chemical/CD22 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/CXCL1 protein, human,
http://linkedlifedata.com/resource/pubmed/chemical/Cell Adhesion Molecules,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL2,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL3,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL4,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL5,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CXCL1,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokines,
http://linkedlifedata.com/resource/pubmed/chemical/Chemokines, CXC,
http://linkedlifedata.com/resource/pubmed/chemical/Chemotactic Factors,
http://linkedlifedata.com/resource/pubmed/chemical/Growth Substances,
http://linkedlifedata.com/resource/pubmed/chemical/Immunoglobulins, Intravenous,
http://linkedlifedata.com/resource/pubmed/chemical/Indium Radioisotopes,
http://linkedlifedata.com/resource/pubmed/chemical/Intercellular Signaling Peptides...,
http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-8,
http://linkedlifedata.com/resource/pubmed/chemical/Lectins,
http://linkedlifedata.com/resource/pubmed/chemical/Macrophage Inflammatory Proteins,
http://linkedlifedata.com/resource/pubmed/chemical/Tumor Necrosis Factor-alpha
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
0004-3591
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
43
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
38-47
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:10643698-Aged,
pubmed-meshheading:10643698-Antibodies, Monoclonal,
pubmed-meshheading:10643698-Antigens, CD,
pubmed-meshheading:10643698-Antigens, CD22,
pubmed-meshheading:10643698-Antigens, CD3,
pubmed-meshheading:10643698-Antigens, Differentiation, B-Lymphocyte,
pubmed-meshheading:10643698-Arthritis, Rheumatoid,
pubmed-meshheading:10643698-B-Lymphocytes,
pubmed-meshheading:10643698-Cell Adhesion Molecules,
pubmed-meshheading:10643698-Cell Movement,
pubmed-meshheading:10643698-Chemokine CCL2,
pubmed-meshheading:10643698-Chemokine CCL3,
pubmed-meshheading:10643698-Chemokine CCL4,
pubmed-meshheading:10643698-Chemokine CCL5,
pubmed-meshheading:10643698-Chemokine CXCL1,
pubmed-meshheading:10643698-Chemokines,
pubmed-meshheading:10643698-Chemokines, CXC,
pubmed-meshheading:10643698-Chemotactic Factors,
pubmed-meshheading:10643698-Female,
pubmed-meshheading:10643698-Growth Substances,
pubmed-meshheading:10643698-Humans,
pubmed-meshheading:10643698-Immunoglobulins, Intravenous,
pubmed-meshheading:10643698-Indium Radioisotopes,
pubmed-meshheading:10643698-Intercellular Signaling Peptides and Proteins,
pubmed-meshheading:10643698-Interleukin-8,
pubmed-meshheading:10643698-Joints,
pubmed-meshheading:10643698-Lectins,
pubmed-meshheading:10643698-Leukocytes,
pubmed-meshheading:10643698-Macrophage Inflammatory Proteins,
pubmed-meshheading:10643698-Male,
pubmed-meshheading:10643698-Middle Aged,
pubmed-meshheading:10643698-Neutrophils,
pubmed-meshheading:10643698-Synovial Fluid,
pubmed-meshheading:10643698-Synovial Membrane,
pubmed-meshheading:10643698-T-Lymphocytes,
pubmed-meshheading:10643698-Tumor Necrosis Factor-alpha
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pubmed:year |
2000
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pubmed:articleTitle |
Reduction of chemokine levels and leukocyte traffic to joints by tumor necrosis factor alpha blockade in patients with rheumatoid arthritis.
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pubmed:affiliation |
Kennedy Institute of Rheumatology, London, UK.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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