rdf:type |
|
lifeskim:mentions |
umls-concept:C0021760,
umls-concept:C0021764,
umls-concept:C0030685,
umls-concept:C0032961,
umls-concept:C0033554,
umls-concept:C0205263,
umls-concept:C0205307,
umls-concept:C0205359,
umls-concept:C0391871,
umls-concept:C0680255,
umls-concept:C0694695,
umls-concept:C1283071,
umls-concept:C1456820,
umls-concept:C1963578
|
pubmed:issue |
6
|
pubmed:dateCreated |
2000-4-18
|
pubmed:abstractText |
The objective of this study was to clarify the role of the main proinflammatory cytokines (interleukin [IL]-1, IL-6, tumor necrosis factor [TNF]-alpha) in the pathogenesis of preeclampsia and how these cytokines affect one another and the production of prostaglandins (PGs).
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Dec
|
pubmed:issn |
1046-7408
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
42
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
369-74
|
pubmed:dateRevised |
2004-11-17
|
pubmed:meshHeading |
|
pubmed:year |
1999
|
pubmed:articleTitle |
Spontaneous and induced release of prostaglandins, interleukin (IL)-1beta, IL-6, and tumor necrosis factor-alpha by placental tissue from normal and preeclamptic pregnancies.
|
pubmed:affiliation |
Department of Internal Medicine, Immunology and Infectious Disease (MIDIM), University of Bari, Italy.
|
pubmed:publicationType |
Journal Article
|