pubmed-article:978585 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C1179517 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C0021641 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C0002712 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C0030685 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C0680255 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C0391871 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C1283071 | lld:lifeskim |
pubmed-article:978585 | lifeskim:mentions | umls-concept:C1963578 | lld:lifeskim |
pubmed-article:978585 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:978585 | pubmed:dateCreated | 1977-1-3 | lld:pubmed |
pubmed-article:978585 | pubmed:abstractText | 1. Insulin (1 mu./ml.) potentiated the release of amylase from isolated pancreas of rats perfused with erythrocyte-containing medium and stimulated by 0-5 m-u. pancreozymin (Pz)/ml., whereas the same concentration of insulin failed to potentiate the response evoked by or 200 m-u. PZ/ml. 2. Intracellular measurement of membrane potentials from the acinar cells of the same preparations showed that insulin (1 mu./ml.) simultaneously potentiates the hyperpolarization and amylase release in response to 0-5 m-u. PZ/ml. 3. These effects of insulin on the PZ-induced responses were inhibited by ouabain (3 X 10(-5) M). 4. The results suggest that insulin, in the concentration studied, has a potentiating action on the activity of an electrogenic Na pump, which maintains the hyperpolarization and amylase release during continuous stimulation with Pz. 5. Insulin in also potentiated the Pz-induced amylase release in the rat pancreata in situ after vagotomy and ligature of the pyloric region. | lld:pubmed |
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pubmed-article:978585 | pubmed:language | eng | lld:pubmed |
pubmed-article:978585 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:978585 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:978585 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:978585 | pubmed:month | Oct | lld:pubmed |
pubmed-article:978585 | pubmed:issn | 0022-3751 | lld:pubmed |
pubmed-article:978585 | pubmed:author | pubmed-author:KannoTT | lld:pubmed |
pubmed-article:978585 | pubmed:author | pubmed-author:SaitoAA | lld:pubmed |
pubmed-article:978585 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:978585 | pubmed:volume | 261 | lld:pubmed |
pubmed-article:978585 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:978585 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:978585 | pubmed:pagination | 505-21 | lld:pubmed |
pubmed-article:978585 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:978585 | pubmed:year | 1976 | lld:pubmed |
pubmed-article:978585 | pubmed:articleTitle | The potentiating influences of insulin on pancreozymin-induced hyperpolarization and amylase release in the pancreatic acinar cell. | lld:pubmed |
pubmed-article:978585 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:978585 | pubmed:publicationType | In Vitro | lld:pubmed |
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