Switch to
| Predicate | Object |
|---|---|
| rdf:type | |
| lifeskim:mentions | |
| pubmed:issue |
2
|
| pubmed:dateCreated |
1998-10-15
|
| pubmed:abstractText |
Ischemia-reperfusion injury after organ transplantation is a major cause of delayed graft function. We showed earlier that antisense oligodesoxynucleotides (ODN) for intercellular adhesion molecule-1 (ICAM-1) ameliorate reperfusion injury after acute ischemia. This study tested the hypothesis that antisense ODN for ICAM-1 prevents ischemia-reperfusion injury and facilitates immediate graft function in a rat autotransplantation model.
|
| pubmed:language |
eng
|
| pubmed:journal | |
| pubmed:citationSubset |
IM
|
| pubmed:chemical | |
| pubmed:status |
MEDLINE
|
| pubmed:month |
Aug
|
| pubmed:issn |
0085-2538
|
| pubmed:author | |
| pubmed:issnType |
Print
|
| pubmed:volume |
54
|
| pubmed:owner |
NLM
|
| pubmed:authorsComplete |
Y
|
| pubmed:pagination |
590-602
|
| pubmed:dateRevised |
2006-11-15
|
| pubmed:meshHeading |
pubmed-meshheading:9690227-Animals,
pubmed-meshheading:9690227-Intercellular Adhesion Molecule-1,
pubmed-meshheading:9690227-Kidney,
pubmed-meshheading:9690227-Kidney Transplantation,
pubmed-meshheading:9690227-Male,
pubmed-meshheading:9690227-Oligonucleotides, Antisense,
pubmed-meshheading:9690227-Rats,
pubmed-meshheading:9690227-Rats, Inbred Lew,
pubmed-meshheading:9690227-Reperfusion Injury,
pubmed-meshheading:9690227-Transplantation, Autologous
|
| pubmed:year |
1998
|
| pubmed:articleTitle |
ICAM-1 antisense oligodesoxynucleotides prevent reperfusion injury and enhance immediate graft function in renal transplantation.
|
| pubmed:affiliation |
Franz Volhard Clinic, Max Delbrück Center for Molecular Medicine, Humboldt University of Berlin, Germany.
|
| pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|