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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
1998-8-3
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pubmed:abstractText |
Interleukin-8 is thought to play a role in neutrophil activation and transcapillary migration into the interstitium. Because neutrophils are principal effector cells in acute myocardial ischemia-reperfusion injury, we postulated that the inhibition of interleukin-8 activity with a neutralizing monoclonal antibody directed against rabbit interleukin-8 (ARIL8.2) would attenuate the degree of myocardial injury encountered during reperfusion.
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pubmed:grant |
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0022-5223
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
116
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
114-21
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading |
pubmed-meshheading:9671905-Animals,
pubmed-meshheading:9671905-Antibodies, Monoclonal,
pubmed-meshheading:9671905-Blood Pressure,
pubmed-meshheading:9671905-Cell Movement,
pubmed-meshheading:9671905-Disease Models, Animal,
pubmed-meshheading:9671905-Interleukin-8,
pubmed-meshheading:9671905-Myocardial Reperfusion Injury,
pubmed-meshheading:9671905-Neutrophil Activation,
pubmed-meshheading:9671905-Neutrophils,
pubmed-meshheading:9671905-Peroxidase,
pubmed-meshheading:9671905-Rabbits,
pubmed-meshheading:9671905-Regional Blood Flow
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pubmed:year |
1998
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pubmed:articleTitle |
Inhibition of interleukin-8 blocks myocardial ischemia-reperfusion injury.
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pubmed:affiliation |
Department of Surgery, University of Washington, Seattle 98195, USA.
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, U.S. Gov't, P.H.S.,
Research Support, Non-U.S. Gov't
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