| pubmed-article:9641919 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0442504 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0019564 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0080093 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0205360 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C1152805 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0024779 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
| pubmed-article:9641919 | lifeskim:mentions | umls-concept:C0443252 | lld:lifeskim |
| pubmed-article:9641919 | pubmed:issue | 5372 | lld:pubmed |
| pubmed-article:9641919 | pubmed:dateCreated | 1998-7-16 | lld:pubmed |
| pubmed-article:9641919 | pubmed:abstractText | Hippocampal pyramidal cells are called place cells because each cell tends to fire only when the animal is in a particular part of the environment-the cell's firing field. Acute pharmacological blockade of N-methyl-D-aspartate (NMDA) glutamate receptors was used to investigate how NMDA-based synaptic plasticity participates in the formation and maintenance of the firing fields. The results suggest that the formation and short-term stability of firing fields in a new environment involve plasticity that is independent of NMDA receptor activation. By contrast, the long-term stabilization of newly established firing fields required normal NMDA receptor function and, therefore, may be related to other NMDA-dependent processes such as long-term potentiation and spatial learning. | lld:pubmed |
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| pubmed-article:9641919 | pubmed:language | eng | lld:pubmed |
| pubmed-article:9641919 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:9641919 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:9641919 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:9641919 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:9641919 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:ShapiroMM | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:KandelE RER | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:HawkinsR DRD | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:KentrosCC | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:HargreavesEE | lld:pubmed |
| pubmed-article:9641919 | pubmed:author | pubmed-author:MulkeyS SSS | lld:pubmed |
| pubmed-article:9641919 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:9641919 | pubmed:day | 26 | lld:pubmed |
| pubmed-article:9641919 | pubmed:volume | 280 | lld:pubmed |
| pubmed-article:9641919 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:9641919 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:9641919 | pubmed:pagination | 2121-6 | lld:pubmed |
| pubmed-article:9641919 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:9641919 | pubmed:year | 1998 | lld:pubmed |
| pubmed-article:9641919 | pubmed:articleTitle | Abolition of long-term stability of new hippocampal place cell maps by NMDA receptor blockade. | lld:pubmed |
| pubmed-article:9641919 | pubmed:affiliation | Department of Physiology, SUNY Health Science Center Brooklyn, 450 Clarkson Avenue, Brooklyn, NY 11203, USA. | lld:pubmed |
| pubmed-article:9641919 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:9641919 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:9641919 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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