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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
1993-4-6
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pubmed:abstractText |
Induction of stress proteins, such as heat-shock protein 71 (HSP71), is associated with cardioprotection in isolated ischemic myocardium. We tested this hypothesis in rabbits pretreated with whole-body hyperthermia and then subjected to 30 or 45 minutes of regional coronary occlusion (CO) followed by 3 hours reperfusion (Rep).
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
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|
pubmed:month |
Mar
|
|
pubmed:issn |
0009-7322
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
87
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
963-71
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pubmed:dateRevised |
2010-3-24
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pubmed:meshHeading |
pubmed-meshheading:8443915-Animals,
pubmed-meshheading:8443915-Coronary Disease,
pubmed-meshheading:8443915-Energy Metabolism,
pubmed-meshheading:8443915-Fever,
pubmed-meshheading:8443915-Heat-Shock Proteins,
pubmed-meshheading:8443915-Male,
pubmed-meshheading:8443915-Myocardial Reperfusion,
pubmed-meshheading:8443915-Myocardium,
pubmed-meshheading:8443915-Necrosis,
pubmed-meshheading:8443915-Phosphates,
pubmed-meshheading:8443915-Rabbits
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pubmed:year |
1993
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pubmed:articleTitle |
Heat-shock response and limitation of tissue necrosis during occlusion/reperfusion in rabbit hearts.
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pubmed:affiliation |
Department of Anatomy, Faculty of Medicine, Dalhousie University, Halifax.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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