| pubmed-article:8076691 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C0812246 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C0376515 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C0311404 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C1749855 | lld:lifeskim |
| pubmed-article:8076691 | lifeskim:mentions | umls-concept:C1883709 | lld:lifeskim |
| pubmed-article:8076691 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:8076691 | pubmed:dateCreated | 1994-10-3 | lld:pubmed |
| pubmed-article:8076691 | pubmed:abstractText | The recent demonstration of the anti-oxidant properties of the Bcl-2 gene product suggested that expression of Bcl-2 may interfere with the nuclear migration of the NF-kappa B transcription factor, which is thought to depend on the presence of reactive oxygen intermediates. In mouse L cells, overexpression of Bcl-2 interfered with the activation of NF-kappa B by H2O2. However, Bcl-2 had no effect on the activation of NF-kappa B by TNF, even though it protected cells from TNF-induced apoptosis. The effects of exogenous pyrrolidine dithiocarbamate were very similar to those of Bcl-2 overexpression. We conclude that the protective effects of anti-oxidants against induced apoptotic cell death are unrelated to their ability to interfere with NF-kappa B activation. | lld:pubmed |
| pubmed-article:8076691 | pubmed:language | eng | lld:pubmed |
| pubmed-article:8076691 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:8076691 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:8076691 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:8076691 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:8076691 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:8076691 | pubmed:issn | 0014-5793 | lld:pubmed |
| pubmed-article:8076691 | pubmed:author | pubmed-author:AlbrechtHH | lld:pubmed |
| pubmed-article:8076691 | pubmed:author | pubmed-author:JongeneelC... | lld:pubmed |
| pubmed-article:8076691 | pubmed:author | pubmed-author:TschoppJJ | lld:pubmed |
| pubmed-article:8076691 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:8076691 | pubmed:day | 29 | lld:pubmed |
| pubmed-article:8076691 | pubmed:volume | 351 | lld:pubmed |
| pubmed-article:8076691 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:8076691 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:8076691 | pubmed:pagination | 45-8 | lld:pubmed |
| pubmed-article:8076691 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:8076691 | pubmed:meshHeading | pubmed-meshheading:8076691-... | lld:pubmed |
| pubmed-article:8076691 | pubmed:year | 1994 | lld:pubmed |
| pubmed-article:8076691 | pubmed:articleTitle | Bcl-2 protects from oxidative damage and apoptotic cell death without interfering with activation of NF-kappa B by TNF. | lld:pubmed |
| pubmed-article:8076691 | pubmed:affiliation | Ludwig Institute for Cancer Research, University of Lausanne, Epalinges, Switzerland. | lld:pubmed |
| pubmed-article:8076691 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:8076691 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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