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rdf:type |
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|
lifeskim:mentions |
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pubmed:issue |
7
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pubmed:dateCreated |
1994-7-29
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pubmed:abstractText |
Thrombolysis is a promising therapy for acute ischemic stroke. However, there is evidence that neutrophils may physically plug cerebral microvessels on reperfusion, preventing the full benefit of thrombolysis. We undertook this study to determine whether there was increased endothelial expression of the intercellular adhesion molecule-1 (ICAM-1) gene during hypoxia-reoxygenation.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
|
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
0039-2499
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pubmed:author |
|
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pubmed:issnType |
Print
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1463-7; discussion 1468
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:7912865-Brain,
pubmed-meshheading:7912865-Brain Ischemia,
pubmed-meshheading:7912865-Cell Adhesion Molecules,
pubmed-meshheading:7912865-Cell Hypoxia,
pubmed-meshheading:7912865-Cerebrovascular Disorders,
pubmed-meshheading:7912865-Endothelium, Vascular,
pubmed-meshheading:7912865-Gene Expression,
pubmed-meshheading:7912865-Humans,
pubmed-meshheading:7912865-Intercellular Adhesion Molecule-1,
pubmed-meshheading:7912865-Oxygen,
pubmed-meshheading:7912865-Reperfusion Injury,
pubmed-meshheading:7912865-Up-Regulation
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pubmed:year |
1994
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pubmed:articleTitle |
Increased expression of ICAM-1 during reoxygenation in brain endothelial cells.
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pubmed:affiliation |
Department of Neurology, Veterans Affairs Medical Center, Augusta, Ga.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, Non-P.H.S.
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