21410931

Source:http://linkedlifedata.com/resource/pubmed/id/21410931

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0007634, umls-concept:C0174680, umls-concept:C0205225, umls-concept:C0441655, umls-concept:C0851827, umls-concept:C0971285, umls-concept:C1514559, umls-concept:C1550548, umls-concept:C1555714, umls-concept:C1701901, umls-concept:C1705654
pubmed:dateCreated	2011-4-1
pubmed:abstractText	Aurora-A is a bona-fide oncogene whose expression is associated with genomic instability and malignant transformation. In several types of cancer, gene amplification and/or increased protein levels of Aurora-A are a common feature.
pubmed:commentsCorrections	http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-10559918, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11166196, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11259099, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11291073, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11376125, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11847097, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-11976319, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-12208897, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-12631597, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-13678582, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-14523000, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-14644191, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-14702041, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-14743206, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-14993419, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15128871, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15182704, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15327889, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15469940, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15480417, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15545627, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15573114, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-15923640, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17182870, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17182871, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17438137, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17510316, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17563743, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-17604723, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-18801727, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-19132228, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-19435814, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-19441883, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-19668197, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-19887370, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-20461437, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-20482313, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-21087322, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-21102634, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-9054499, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-9190208, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-9606188, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-9771714, http://linkedlifedata.com/resource/pubmed/commentcorrection/21410931-9776764
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101147698
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/CCND1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Cyclin D1, http://linkedlifedata.com/resource/pubmed/chemical/Mutant Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Protein-Serine-Threonine Kinases, http://linkedlifedata.com/resource/pubmed/chemical/RASSF1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Protein p53, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Suppressor Proteins, http://linkedlifedata.com/resource/pubmed/chemical/aurora kinase, http://linkedlifedata.com/resource/pubmed/chemical/ras GTPase-Activating Proteins
pubmed:status	MEDLINE
pubmed:issn	1476-4598
pubmed:author	pubmed-author:BergerWalterW, pubmed-author:JantscherFlorianF, pubmed-author:MayerChristoph-ErikCE, pubmed-author:PirkerChristineC, pubmed-author:SutterluetyHedwigH
pubmed:issnType	Electronic
pubmed:volume	10
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	28
pubmed:dateRevised	2011-7-27
pubmed:meshHeading	pubmed-meshheading:21410931-Cell Proliferation, pubmed-meshheading:21410931-Cells, Cultured, pubmed-meshheading:21410931-Cyclin D1, pubmed-meshheading:21410931-G0 Phase, pubmed-meshheading:21410931-G1 Phase, pubmed-meshheading:21410931-Gene Expression Regulation, pubmed-meshheading:21410931-Humans, pubmed-meshheading:21410931-Mitosis, pubmed-meshheading:21410931-Mutant Proteins, pubmed-meshheading:21410931-Protein-Serine-Threonine Kinases, pubmed-meshheading:21410931-S Phase, pubmed-meshheading:21410931-Time Factors, pubmed-meshheading:21410931-Tumor Suppressor Protein p53, pubmed-meshheading:21410931-Tumor Suppressor Proteins, pubmed-meshheading:21410931-ras GTPase-Activating Proteins
pubmed:year	2011
pubmed:articleTitle	Overexpression of Aurora-A in primary cells interferes with S-phase entry by diminishing Cyclin D1 dependent activities.

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