pubmed-article:20877570 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C1332717 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C1706438 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0085358 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0054871 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0205734 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C1413244 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C2698600 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C0301625 | lld:lifeskim |
pubmed-article:20877570 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
pubmed-article:20877570 | pubmed:issue | 9 | lld:pubmed |
pubmed-article:20877570 | pubmed:dateCreated | 2010-9-29 | lld:pubmed |
pubmed-article:20877570 | pubmed:abstractText | Type 1 diabetes (T1D) is an autoimmune disease resulting from defects in central and peripheral tolerance and characterized by T cell-mediated destruction of islet ? cells. To determine whether specific lysosomal proteases might influence the outcome of a T cell-mediated autoimmune response, we examined the functional significance of cathepsin inhibition on autoimmune T1D-prone non-obese diabetic (NOD) mice. | lld:pubmed |
pubmed-article:20877570 | pubmed:language | eng | lld:pubmed |
pubmed-article:20877570 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20877570 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:20877570 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20877570 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20877570 | pubmed:issn | 1932-6203 | lld:pubmed |
pubmed-article:20877570 | pubmed:author | pubmed-author:KatunumaNobuh... | lld:pubmed |
pubmed-article:20877570 | pubmed:author | pubmed-author:YamadaAkikoA | lld:pubmed |
pubmed-article:20877570 | pubmed:author | pubmed-author:HayashiYoshio... | lld:pubmed |
pubmed-article:20877570 | pubmed:author | pubmed-author:IshimaruNaozu... | lld:pubmed |
pubmed-article:20877570 | pubmed:author | pubmed-author:ArakakiRiekoR | lld:pubmed |
pubmed-article:20877570 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20877570 | pubmed:volume | 5 | lld:pubmed |
pubmed-article:20877570 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20877570 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20877570 | pubmed:pagination | e12894 | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
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pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:meshHeading | pubmed-meshheading:20877570... | lld:pubmed |
pubmed-article:20877570 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20877570 | pubmed:articleTitle | Cathepsin L inhibition prevents murine autoimmune diabetes via suppression of CD8(+) T cell activity. | lld:pubmed |
pubmed-article:20877570 | pubmed:affiliation | Department of Oral Molecular Pathology, Institute of Health Biosciences, The University of Tokushima Graduate School, Tokushima, Japan. | lld:pubmed |
pubmed-article:20877570 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20877570 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
entrez-gene:13039 | entrezgene:pubmed | pubmed-article:20877570 | lld:entrezgene |
http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:20877570 | lld:entrezgene |