Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
12
pubmed:dateCreated
2010-12-1
pubmed:abstractText
Deficiency of ?(1) -antitrypsin (?(1) AT) may be a determinant of susceptibility to Wegener's granulomatosis (WG). Several previous, mainly small, case-control studies have shown that 5-27% of patients with WG carried the ?(1) AT deficiency Z allele. It is not clear whether the S allele, the other major ?(1) AT deficiency variant, is associated with WG. This study investigated the relationship of the ?(1) AT deficiency Z and S alleles with the risk of developing WG in a large cohort.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
1529-0131
pubmed:author
pubmed:copyrightInfo
Copyright © 2010 by the American College of Rheumatology.
pubmed:issnType
Electronic
pubmed:volume
62
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
3760-7
pubmed:dateRevised
2011-9-26
pubmed:meshHeading
pubmed:year
2010
pubmed:articleTitle
Alpha?-antitrypsin deficiency-related alleles Z and S and the risk of Wegener's granulomatosis.
pubmed:affiliation
Boston University School of Medicine, Boston, Massachusetts, USA.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural