Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
11
pubmed:dateCreated
2010-9-6
pubmed:abstractText
Peroxiredoxins are redox-sensing enzymes with multiple cellular functions. Previously, we reported on the potent antioxidant function of Drosophila peroxiredoxin 5 (dPrx5). Studies with mammalian and human cells suggest that peroxiredoxins can modulate immune-related signaling.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
0006-3002
pubmed:author
pubmed:copyrightInfo
Copyright © 2010 Elsevier Inc. All rights reserved.
pubmed:issnType
Print
pubmed:volume
1800
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1153-63
pubmed:dateRevised
2011-11-1
pubmed:meshHeading
pubmed-meshheading:20600624-Animals, pubmed-meshheading:20600624-Beauveria, pubmed-meshheading:20600624-Blotting, Western, pubmed-meshheading:20600624-Cells, Cultured, pubmed-meshheading:20600624-Drosophila melanogaster, pubmed-meshheading:20600624-Epistasis, Genetic, pubmed-meshheading:20600624-Humans, pubmed-meshheading:20600624-Immunity, Innate, pubmed-meshheading:20600624-Insect Proteins, pubmed-meshheading:20600624-Longevity, pubmed-meshheading:20600624-MAP Kinase Kinase 4, pubmed-meshheading:20600624-Mutation, pubmed-meshheading:20600624-Oxidative Stress, pubmed-meshheading:20600624-Peroxiredoxins, pubmed-meshheading:20600624-Phenotype, pubmed-meshheading:20600624-Phosphorylation, pubmed-meshheading:20600624-RNA, Messenger, pubmed-meshheading:20600624-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:20600624-Sepsis, pubmed-meshheading:20600624-Signal Transduction
pubmed:year
2010
pubmed:articleTitle
Peroxiredoxin 5 modulates immune response in Drosophila.
pubmed:affiliation
Department of Biological Sciences, Southern Methodist University, 6501 Airline Rd., Room 113, Dallas, TX 75275, USA. snradyuk@smu.edu
pubmed:publicationType
Journal Article, Comparative Study, Research Support, N.I.H., Extramural