Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2010-3-22
pubmed:abstractText
Although interleukin-17 (IL-17) has been reported to participate in the pathogenesis of infectious, autoimmune and allergic disorders, the precise role in allograft rejection remains uncertain. This study illustrates that IL-17 contributes to the pathogenesis of chronic allograft rejection.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
1573-2592
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
30
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
235-40
pubmed:meshHeading
pubmed-meshheading:20130970-Animals, pubmed-meshheading:20130970-Antigens, CD, pubmed-meshheading:20130970-Cell Separation, pubmed-meshheading:20130970-Cells, Cultured, pubmed-meshheading:20130970-Chronic Disease, pubmed-meshheading:20130970-Flow Cytometry, pubmed-meshheading:20130970-Graft Occlusion, Vascular, pubmed-meshheading:20130970-Graft Rejection, pubmed-meshheading:20130970-Heart Transplantation, pubmed-meshheading:20130970-Interleukin-17, pubmed-meshheading:20130970-Mice, pubmed-meshheading:20130970-Mice, Inbred C57BL, pubmed-meshheading:20130970-Mice, Knockout, pubmed-meshheading:20130970-Mice, Mutant Strains, pubmed-meshheading:20130970-Myocardium, pubmed-meshheading:20130970-Receptors, Antigen, T-Cell, gamma-delta, pubmed-meshheading:20130970-T-Lymphocyte Subsets, pubmed-meshheading:20130970-T-Lymphocytes
pubmed:year
2010
pubmed:articleTitle
IL-17 contributes to the development of chronic rejection in a murine heart transplant model.
pubmed:affiliation
Department of Cardiothoracic Surgery, Stanford University School of Medicine, Stanford, CA 94305, USA.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't