| pubmed-article:1934069 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:1934069 | lifeskim:mentions | umls-concept:C0013138 | lld:lifeskim |
| pubmed-article:1934069 | lifeskim:mentions | umls-concept:C2335126 | lld:lifeskim |
| pubmed-article:1934069 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:1934069 | lifeskim:mentions | umls-concept:C1334043 | lld:lifeskim |
| pubmed-article:1934069 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:1934069 | pubmed:dateCreated | 1991-12-26 | lld:pubmed |
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| pubmed-article:1934069 | pubmed:abstractText | Roughened is a dominant mutation of D. melanogaster that disrupts eye development. The majority of the ommatidia in the adult eye lack a single photoreceptor cell, which is most commonly the R7 cell. The Roughened mutation disrupts the early stages of photoreceptor cell determination. Roughened is a dominant gain-of-function mutation that results from a single amino acid change (Phe157 to Leu) in the Drosophila Rap1 protein. Loss of function Rap1 mutations are lethal. Drosophila Rap1 protein is 88% identical to human rap1A/K-rev1 protein, a putative antagonist of ras action. | lld:pubmed |
| pubmed-article:1934069 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1934069 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1934069 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1934069 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1934069 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:1934069 | pubmed:issn | 0092-8674 | lld:pubmed |
| pubmed-article:1934069 | pubmed:author | pubmed-author:RubinG MGM | lld:pubmed |
| pubmed-article:1934069 | pubmed:author | pubmed-author:CarthewR WRW | lld:pubmed |
| pubmed-article:1934069 | pubmed:author | pubmed-author:HariharanI... | lld:pubmed |
| pubmed-article:1934069 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1934069 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:1934069 | pubmed:volume | 67 | lld:pubmed |
| pubmed-article:1934069 | pubmed:geneSymbol | R | lld:pubmed |
| pubmed-article:1934069 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1934069 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1934069 | pubmed:pagination | 717-22 | lld:pubmed |
| pubmed-article:1934069 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:1934069 | pubmed:year | 1991 | lld:pubmed |
| pubmed-article:1934069 | pubmed:articleTitle | The Drosophila roughened mutation: activation of a rap homolog disrupts eye development and interferes with cell determination. | lld:pubmed |
| pubmed-article:1934069 | pubmed:affiliation | Howard Hughes Medical Institute, Department of Molecular and Cell Biology, University of California, Berkeley 94720. | lld:pubmed |
| pubmed-article:1934069 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1934069 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:1934069 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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