| pubmed-article:19195504 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0763282 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0014792 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0155626 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0042874 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:19195504 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:19195504 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:19195504 | pubmed:dateCreated | 2009-2-6 | lld:pubmed |
| pubmed-article:19195504 | pubmed:abstractText | Glutathione peroxidase 1 (GPX-1) has an important role in antioxidant defense and has been suggested to have a protective role against the pathogenesis of atherosclerosis. In the present study, erythrocytic GPX-1 activity from 42 patients with acute myocardial infarction and 285 healthy control subjects of Taiwanese origin were determined. Our data showed that mean GPX-1 activity decreased in patients with acute myocardial infarction (40.7 +/- 9.2 vs 47.3 +/- 16.6 U/g hemoglobin; p <0.001). There was a significant inverse correlation between GPX-1 activity and acute myocardial infarction incidence (p = 0.032). In addition, plasma vitamin E inversely correlated with acute myocardial infarction incidence (p = 0.017). The incidence of acute myocardial infarction increased only in patients with low plasma vitamin E and low erythrocytic GPX-1 activity, but not in those with high vitamin E and low GPX-1 activity, low vitamin E and high GPX-1 activity, or high vitamin E and high GPX-1 activity. In conclusion, acute myocardial infarction incidence significantly increased in patients with a concomitant decrease in plasma vitamin E and erythrocytic GPX-1 activity, suggesting that both GPX-1 and vitamin E may be valuable markers for monitoring cardiovascular events, particularly when used in combination. | lld:pubmed |
| pubmed-article:19195504 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19195504 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19195504 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:19195504 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19195504 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19195504 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19195504 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19195504 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:19195504 | pubmed:issn | 1879-1913 | lld:pubmed |
| pubmed-article:19195504 | pubmed:author | pubmed-author:ChengMei-ling... | lld:pubmed |
| pubmed-article:19195504 | pubmed:author | pubmed-author:HoHung-yaoHY | lld:pubmed |
| pubmed-article:19195504 | pubmed:author | pubmed-author:ChiuDaniel... | lld:pubmed |
| pubmed-article:19195504 | pubmed:author | pubmed-author:ChenChin-ming... | lld:pubmed |
| pubmed-article:19195504 | pubmed:author | pubmed-author:LiJui-mingJM | lld:pubmed |
| pubmed-article:19195504 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19195504 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:19195504 | pubmed:volume | 103 | lld:pubmed |
| pubmed-article:19195504 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19195504 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19195504 | pubmed:pagination | 471-5 | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:meshHeading | pubmed-meshheading:19195504... | lld:pubmed |
| pubmed-article:19195504 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19195504 | pubmed:articleTitle | Effect of acute myocardial infarction on erythrocytic glutathione peroxidase 1 activity and plasma vitamin e levels. | lld:pubmed |
| pubmed-article:19195504 | pubmed:affiliation | Graduate Institute of Medical Biotechnology, College of Medicine, Chang Gung University, Kwei-san, Tao-yuan, Taipei, Taiwan. | lld:pubmed |
| pubmed-article:19195504 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19195504 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:2876 | entrezgene:pubmed | pubmed-article:19195504 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:19195504 | lld:entrezgene |
| lhgdn:association:153 | lhgdn:found_in | pubmed-article:19195504 | lld:lhgdn |
