18628478

Source:http://linkedlifedata.com/resource/pubmed/id/18628478

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0015272, umls-concept:C0059239, umls-concept:C0140080, umls-concept:C0279628, umls-concept:C1268567, umls-concept:C1280500, umls-concept:C1515655, umls-concept:C1533691, umls-concept:C1554184, umls-concept:C1565434
pubmed:issue	14
pubmed:dateCreated	2008-7-16
pubmed:abstractText	Focal adhesion kinase (FAK) regulates integrin and growth factor-mediated signaling pathways to enhance cell migration, proliferation, and survival, and its up-regulation correlates malignant grade and poor outcome in several types of cancer. In this study, we aimed to raise a potential therapeutic strategy using a FAK inhibitor for Barrett's esophageal adenocarcinoma.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9502500
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Antineoplastic Agents, http://linkedlifedata.com/resource/pubmed/chemical/Enzyme Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/Focal Adhesion Protein-Tyrosine..., http://linkedlifedata.com/resource/pubmed/chemical/Morpholines, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, IGF Type 1, http://linkedlifedata.com/resource/pubmed/chemical/TAE226
pubmed:status	MEDLINE
pubmed:month	Jul
pubmed:issn	1078-0432
pubmed:author	pubmed-author:BeerDavid GDG, pubmed-author:HaisaMinoruM, pubmed-author:HatakeyamaShinjiS, pubmed-author:MatsuokaJunjiJ, pubmed-author:MotokiTakayukiT, pubmed-author:NagatsukaHitoshiH, pubmed-author:NaomotoYoshioY, pubmed-author:OhmoriOsamuO, pubmed-author:SakuramaKazufumiK, pubmed-author:ShirakawaYasuhiroY, pubmed-author:TakaokaMunenoriM, pubmed-author:TanakaNoriakiN, pubmed-author:TomonoYasukoY, pubmed-author:WatanabeNobuyukiN, pubmed-author:YamatsujiTomokiT
pubmed:issnType	Print
pubmed:day	15
pubmed:volume	14
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	4631-9
pubmed:dateRevised	2009-11-19
pubmed:meshHeading	pubmed-meshheading:18628478-Adenocarcinoma, pubmed-meshheading:18628478-Animals, pubmed-meshheading:18628478-Antineoplastic Agents, pubmed-meshheading:18628478-Apoptosis, pubmed-meshheading:18628478-Blotting, Western, pubmed-meshheading:18628478-Cell Movement, pubmed-meshheading:18628478-Cells, Cultured, pubmed-meshheading:18628478-Enzyme Inhibitors, pubmed-meshheading:18628478-Esophageal Neoplasms, pubmed-meshheading:18628478-Flow Cytometry, pubmed-meshheading:18628478-Fluorescent Antibody Technique, pubmed-meshheading:18628478-Focal Adhesion Protein-Tyrosine Kinases, pubmed-meshheading:18628478-Humans, pubmed-meshheading:18628478-Immunohistochemistry, pubmed-meshheading:18628478-In Situ Nick-End Labeling, pubmed-meshheading:18628478-Mice, pubmed-meshheading:18628478-Morpholines, pubmed-meshheading:18628478-Receptor, IGF Type 1, pubmed-meshheading:18628478-Signal Transduction
pubmed:year	2008
pubmed:articleTitle	Dual tyrosine kinase inhibitor for focal adhesion kinase and insulin-like growth factor-I receptor exhibits anticancer effect in esophageal adenocarcinoma in vitro and in vivo.
pubmed:affiliation	Department of Gastroenterological Surgery, Okayama University, Okayama, Japan.
pubmed:publicationType	Journal Article