Source:http://linkedlifedata.com/resource/pubmed/id/18367112
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
6
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pubmed:dateCreated |
2008-11-10
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pubmed:abstractText |
Cyclooxygenase 2 (COX-2) is aberrantly expressed in multiple tumor types including bladder cancer and is associated with enhanced growth, resistance to apoptosis, invasion, and angiogenesis. To evaluate the mechanisms through which COX-2 expression alters normal urothelium, we transfected the SV-40 immortalized human urothelial cell line SV-HUC with COX-2.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
1078-1439
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
26
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
641-5
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pubmed:meshHeading |
pubmed-meshheading:18367112-Cell Line,
pubmed-meshheading:18367112-Cyclooxygenase 2,
pubmed-meshheading:18367112-Dinoprostone,
pubmed-meshheading:18367112-Humans,
pubmed-meshheading:18367112-Neoplasm Invasiveness,
pubmed-meshheading:18367112-Transfection,
pubmed-meshheading:18367112-Urinary Bladder,
pubmed-meshheading:18367112-Urinary Bladder Neoplasms
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pubmed:articleTitle |
Forced COX-2 expression induces PGE(2) and invasion in immortalized urothelial cells.
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pubmed:affiliation |
William S. Middleton Memorial Veterans Hospital, and Division of Urology, University of Wisconsin Hospital and Clinics, Madison, WI 53705, USA. gee@surgery.wisc.edu
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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