| pubmed-article:18337659 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C0063695 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C0079717 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C1704675 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C0887936 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:18337659 | lifeskim:mentions | umls-concept:C1292733 | lld:lifeskim |
| pubmed-article:18337659 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:18337659 | pubmed:dateCreated | 2008-3-13 | lld:pubmed |
| pubmed-article:18337659 | pubmed:abstractText | Allogeneic tolerance can be reliably obtained with monoclonal antibody therapy targeting CD45RB. Although regulatory T cells play an important role in the mechanism, we have recently demonstrated the active participation of host B lymphocytes. After anti-CD45RB therapy, B lymphocytes demonstrate phenotypic alterations that include up-regulation of CD54 (intercellular adhesion molecule [ICAM]-1). We have investigated the hypothesis that alteration in ICAM-1 expression is required for tolerance induction. | lld:pubmed |
| pubmed-article:18337659 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18337659 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:18337659 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:18337659 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18337659 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18337659 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:18337659 | pubmed:issn | 0041-1337 | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:WangJingJ | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:MarkmannJames... | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:MooreDaniel... | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:YehHeidiH | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:KimJames IJI | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:GuYiY | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:LianMoh-MohMM | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:DengShaopingS | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:HuangXiaolunX | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:MohiuddinMoha... | lld:pubmed |
| pubmed-article:18337659 | pubmed:author | pubmed-author:SonawaneSamsh... | lld:pubmed |
| pubmed-article:18337659 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18337659 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:18337659 | pubmed:volume | 85 | lld:pubmed |
| pubmed-article:18337659 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18337659 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18337659 | pubmed:pagination | 675-80 | lld:pubmed |
| pubmed-article:18337659 | pubmed:dateRevised | 2011-5-17 | lld:pubmed |
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| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:meshHeading | pubmed-meshheading:18337659... | lld:pubmed |
| pubmed-article:18337659 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18337659 | pubmed:articleTitle | Inhibition of ICAM-1/LFA-1 interactions prevents B-cell-dependent anti-CD45RB-induced transplantation tolerance. | lld:pubmed |
| pubmed-article:18337659 | pubmed:affiliation | Department of Surgery, University of Pennsylvania School of Medicine, Philadelphia, Pennsylvania, USA. | lld:pubmed |
| pubmed-article:18337659 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18337659 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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