| pubmed-article:17673218 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C0024375 | lld:lifeskim |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C0013081 | lld:lifeskim |
| pubmed-article:17673218 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:17673218 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:17673218 | pubmed:dateCreated | 2008-2-1 | lld:pubmed |
| pubmed-article:17673218 | pubmed:abstractText | TNFalpha is a pro-inflammatory cytokine that induces endothelial dysfunction and promotes atherosclerosis progression. Down-regulation of lysyl oxidase (LOX), a key enzyme in extracellular matrix maturation, by pro-atherogenic risk factors such as LDL and homocysteine, is associated with an impairment of endothelial barrier function. Our hypothesis is that the inflammatory cytokine TNFalpha could also modulate LOX expression/function in endothelial cells. | lld:pubmed |
| pubmed-article:17673218 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17673218 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:17673218 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17673218 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17673218 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:17673218 | pubmed:issn | 1879-1484 | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:NavarroM AMA | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:RodríguezCC | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:BadimonLL | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:Martínez-Gonz... | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:RaposoBB | lld:pubmed |
| pubmed-article:17673218 | pubmed:author | pubmed-author:AlcudiaJ FJF | lld:pubmed |
| pubmed-article:17673218 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17673218 | pubmed:volume | 196 | lld:pubmed |
| pubmed-article:17673218 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17673218 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17673218 | pubmed:pagination | 558-64 | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:meshHeading | pubmed-meshheading:17673218... | lld:pubmed |
| pubmed-article:17673218 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:17673218 | pubmed:articleTitle | Lysyl oxidase (LOX) down-regulation by TNFalpha: a new mechanism underlying TNFalpha-induced endothelial dysfunction. | lld:pubmed |
| pubmed-article:17673218 | pubmed:affiliation | Centro de Investigación Cardiovascular, CSIC-ICCC, Hospital de Santa Creu i Sant Pau, Barcelona, Spain. | lld:pubmed |
| pubmed-article:17673218 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17673218 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| entrez-gene:4015 | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| entrez-gene:24835 | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| entrez-gene:24914 | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:17673218 | lld:entrezgene |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:17673218 | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:17673218 | lld:pubmed |
