|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0001629,
umls-concept:C0021308,
umls-concept:C0025148,
umls-concept:C0030193,
umls-concept:C0205093,
umls-concept:C0205098,
umls-concept:C0205099,
umls-concept:C0205156,
umls-concept:C0436331,
umls-concept:C1314792,
umls-concept:C1444749,
umls-concept:C1550278
|
|
pubmed:issue |
1
|
|
pubmed:dateCreated |
2007-7-13
|
|
pubmed:abstractText |
Although the pathogenesis of central poststroke pain (CPSP) remains obscure, interactions between the spinothalamic tract and lemniscal sensory tract may be involved in its pathogenic mechanism.
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:status |
MEDLINE
|
|
pubmed:issn |
1421-9913
|
|
pubmed:author |
|
|
pubmed:copyrightInfo |
Copyright 2007 S. Karger AG, Basel.
|
|
pubmed:issnType |
Electronic
|
|
pubmed:volume |
58
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
41-3
|
|
pubmed:meshHeading |
|
|
pubmed:year |
2007
|
|
pubmed:articleTitle |
Medial medullary infarct aggravates central poststroke pain caused by previous lateral medullary infarct.
|
|
pubmed:affiliation |
Department of Neurology, University of Ulsan, Asan Medical Center, Seoul, South Korea. jongskim@amc.seoul.kr
|
|
pubmed:publicationType |
Journal Article,
Case Reports,
Research Support, Non-U.S. Gov't
|
