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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2007-3-5
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pubmed:abstractText |
Activated granulocytes and inflammatory mediators of the innate immune response play fundamental roles in the pathogenesis of acute pancreatitis. We studied whether polymorphisms of interleukin-8 (IL-8) and Toll-like receptor 4 (TLR4) genes correlate with the severity of acute pancreatitis.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
1424-3903
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2006 S. Karger AG, Basel and IAP.
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pubmed:issnType |
Print
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pubmed:volume |
6
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
542-8
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pubmed:meshHeading |
pubmed-meshheading:17124436-Acute Disease,
pubmed-meshheading:17124436-DNA,
pubmed-meshheading:17124436-Genetic Predisposition to Disease,
pubmed-meshheading:17124436-Genotype,
pubmed-meshheading:17124436-Humans,
pubmed-meshheading:17124436-Hungary,
pubmed-meshheading:17124436-Interleukin-5,
pubmed-meshheading:17124436-Pancreatitis,
pubmed-meshheading:17124436-Polymorphism, Single Nucleotide,
pubmed-meshheading:17124436-Prospective Studies,
pubmed-meshheading:17124436-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:17124436-Toll-Like Receptor 4
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pubmed:year |
2006
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pubmed:articleTitle |
Polymorphism in the IL-8 gene, but not in the TLR4 gene, increases the severity of acute pancreatitis.
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pubmed:affiliation |
Department of Medical Microbiology and Immunology, Faculty of Medicine, University of Szeged, Szeged, Hungary.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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