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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
10
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pubmed:dateCreated |
2006-1-2
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pubmed:abstractText |
Chronic allograft nephropathy is the main cause of graft loss. Although many factors are involved in its development, ischemia-reperfusion injury has received increasing attention as a risk determinant. In a previous study of syngeneic renal transplantation and ischemia, we demonstrated a protective effect of acute damage by FTY 720 and antisense oligonucleotides of ICAM-1 (Oligos). The purpose of the current study was to evaluate the impact of these agents on the development of chronic graft damage in the same model.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Dec
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pubmed:issn |
0041-1345
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
37
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
4284-8
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pubmed:dateRevised |
2008-6-5
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pubmed:meshHeading |
pubmed-meshheading:16387098-Animals,
pubmed-meshheading:16387098-Disease Models, Animal,
pubmed-meshheading:16387098-Intercellular Adhesion Molecule-1,
pubmed-meshheading:16387098-Inulin,
pubmed-meshheading:16387098-Kidney,
pubmed-meshheading:16387098-Kidney Function Tests,
pubmed-meshheading:16387098-Male,
pubmed-meshheading:16387098-Oligonucleotides, Antisense,
pubmed-meshheading:16387098-Propylene Glycols,
pubmed-meshheading:16387098-Rats,
pubmed-meshheading:16387098-Rats, Inbred Lew,
pubmed-meshheading:16387098-Reperfusion Injury,
pubmed-meshheading:16387098-Sphingosine
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pubmed:year |
2005
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pubmed:articleTitle |
Use of FTY 720 and ICAM-1 antisense oligonucleotides for attenuating chronic renal damage secondary to ischemia-reperfusion injury.
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pubmed:affiliation |
Unidad de Trasplante Renal, Pontificia Universidad Católica de Chile, Santiago, Chile. pabtron@med.puc.cl
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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