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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
9
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pubmed:dateCreated |
2005-9-6
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pubmed:abstractText |
Rapid hemodynamic deterioration is caused by induction of brain death, but the exact mechanism is still uncertain. The aim of this study was to investigate the contribution of endothelin-1 by using endothelin-1 receptor antagonist (TAK-044) in a canine brain-death model.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Sep
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pubmed:issn |
1557-3117
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
24
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1354-61
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pubmed:dateRevised |
2007-11-15
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pubmed:meshHeading |
pubmed-meshheading:16143257-Animals,
pubmed-meshheading:16143257-Brain Death,
pubmed-meshheading:16143257-Cardiovascular Agents,
pubmed-meshheading:16143257-Coronary Circulation,
pubmed-meshheading:16143257-Dogs,
pubmed-meshheading:16143257-Endothelin-1,
pubmed-meshheading:16143257-Epinephrine,
pubmed-meshheading:16143257-Hemodynamics,
pubmed-meshheading:16143257-Norepinephrine,
pubmed-meshheading:16143257-Peptides, Cyclic,
pubmed-meshheading:16143257-Receptors, Endothelin,
pubmed-meshheading:16143257-Ventricular Dysfunction, Left,
pubmed-meshheading:16143257-Ventricular Function, Left
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pubmed:year |
2005
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pubmed:articleTitle |
Endothelin-1 receptor antagonist prevents deterioration of left ventricular function and coronary flow reserve in brain-dead canine heart.
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pubmed:affiliation |
Department of Cardiovascular Surgery, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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