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| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0206745 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0003595 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:15345516 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:15345516 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:15345516 | pubmed:dateCreated | 2004-11-5 | lld:pubmed |
| pubmed-article:15345516 | pubmed:abstractText | Inflammation plays an important role in atherosclerosis. One of the most potent pro-inflammatory cytokines is tumor necrosis factor-alpha (TNF-alpha), a cytokine identified to have a pathogenic role in chronic inflammatory diseases such as rheumatoid arthritis (RA). The aim of the study was to evaluate the importance of TNF-alpha in atherogenesis. | lld:pubmed |
| pubmed-article:15345516 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15345516 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15345516 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15345516 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:15345516 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:BrånénLenaL | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:NilssonJanJ | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:BengtssonEvaE | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:JovingeStefan... | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:HovgaardLarsL | lld:pubmed |
| pubmed-article:15345516 | pubmed:author | pubmed-author:NitulescuMiha... | lld:pubmed |
| pubmed-article:15345516 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15345516 | pubmed:volume | 24 | lld:pubmed |
| pubmed-article:15345516 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15345516 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15345516 | pubmed:pagination | 2137-42 | lld:pubmed |
| pubmed-article:15345516 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:15345516 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15345516 | pubmed:articleTitle | Inhibition of tumor necrosis factor-alpha reduces atherosclerosis in apolipoprotein E knockout mice. | lld:pubmed |
| pubmed-article:15345516 | pubmed:affiliation | Department of Medicine, University Hospital MAS, Experimental Cardiovascular Research, Wallenberg Laboratory, Ing 46, Plan 1, S-205 02 Malmö, Sweden. lena.branen@medforsk.mas.lu.se | lld:pubmed |
| pubmed-article:15345516 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15345516 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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