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| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0225336 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0063695 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0078056 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:14741380 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:14741380 | pubmed:issue | 1-3 | lld:pubmed |
| pubmed-article:14741380 | pubmed:dateCreated | 2004-1-26 | lld:pubmed |
| pubmed-article:14741380 | pubmed:abstractText | Retinoic acid receptor-related orphan receptor-alpha (RORalpha) is a nuclear orphan receptor. Adenovirus-mediated overexpression of RORalpha1 and RORalpha4 suppressed tumor necrosis factor-alpha (TNF-alpha)-induced expression of vascular cell adhesion molecule-1 (VCAM-1) and intracellular adhesion molecule-1 (ICAM-1) in human umbilical vein endothelial cells. Overexpression of RORalpha1 and RORalpha4 also suppressed TNF-alpha-stimulated translocation of p50 and p65 to the nucleus. In contrast, dominant-negative deletion mutants of RORalpha1 and RORalpha4 failed to suppress the induction of VCAM-1 and ICAM-1 and translocations of p50 and p65. These results suggest that RORalpha1 and RORalpha4 regulate the inflammatory responses via inhibition of the nuclear factor-kappaB signaling pathway in endothelial cells. | lld:pubmed |
| pubmed-article:14741380 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:14741380 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:14741380 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14741380 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:14741380 | pubmed:issn | 0014-5793 | lld:pubmed |
| pubmed-article:14741380 | pubmed:author | pubmed-author:MorserJohnJ | lld:pubmed |
| pubmed-article:14741380 | pubmed:author | pubmed-author:SatozawaNobor... | lld:pubmed |
| pubmed-article:14741380 | pubmed:author | pubmed-author:MigitaHideyuk... | lld:pubmed |
| pubmed-article:14741380 | pubmed:author | pubmed-author:LinJiing-Huey... | lld:pubmed |
| pubmed-article:14741380 | pubmed:author | pubmed-author:KawaiKohichiK | lld:pubmed |
| pubmed-article:14741380 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14741380 | pubmed:day | 16 | lld:pubmed |
| pubmed-article:14741380 | pubmed:volume | 557 | lld:pubmed |
| pubmed-article:14741380 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14741380 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14741380 | pubmed:pagination | 269-74 | lld:pubmed |
| pubmed-article:14741380 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:14741380 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:14741380 | pubmed:articleTitle | RORalpha1 and RORalpha4 suppress TNF-alpha-induced VCAM-1 and ICAM-1 expression in human endothelial cells. | lld:pubmed |
| pubmed-article:14741380 | pubmed:affiliation | Cardiovascular Research, Drug Discovery Institute, Nihon Schering KK, 1900-1, Togo Mobara, Chiba 297-0017, Japan. migitabb@yahoo.co.jp | lld:pubmed |
| pubmed-article:14741380 | pubmed:publicationType | Journal Article | lld:pubmed |
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