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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
3
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pubmed:dateCreated |
2003-8-18
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pubmed:abstractText |
Retinoic acids, derivatives of vitamin A, act through retinoid receptors that are expressed in renal and immunocompetent cells (B and T cells; monocytes and macrophages). In experimental models of glomerulonephritis and renal interstitial disease, retinoids were shown to reduce both glomerular and tubular damage and inflammation. We therefore examined whether retinoids reduce cellular rejection and renal damage in a model of acute renal allograft rejection.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0041-1337
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:day |
15
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pubmed:volume |
76
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
480-9
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:12923432-Albuminuria,
pubmed-meshheading:12923432-Animals,
pubmed-meshheading:12923432-Creatine,
pubmed-meshheading:12923432-Graft Rejection,
pubmed-meshheading:12923432-Immunohistochemistry,
pubmed-meshheading:12923432-Isotretinoin,
pubmed-meshheading:12923432-Kidney,
pubmed-meshheading:12923432-Kidney Glomerulus,
pubmed-meshheading:12923432-Kidney Transplantation,
pubmed-meshheading:12923432-Male,
pubmed-meshheading:12923432-Rats,
pubmed-meshheading:12923432-Rats, Inbred F344,
pubmed-meshheading:12923432-Rats, Inbred Lew
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pubmed:year |
2003
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pubmed:articleTitle |
Isotretinoin ameliorates renal damage in experimental acute renal allograft rejection.
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pubmed:affiliation |
Department of Cellular and Molecular Pathology, German Cancer Research Center, Heidelberg, Germany.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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