rdf:type |
|
lifeskim:mentions |
umls-concept:C0003873,
umls-concept:C0016030,
umls-concept:C0017262,
umls-concept:C0023516,
umls-concept:C0024432,
umls-concept:C0063695,
umls-concept:C0333348,
umls-concept:C0441712,
umls-concept:C1171362,
umls-concept:C1515670,
umls-concept:C1550315,
umls-concept:C1704675,
umls-concept:C1879547
|
pubmed:issue |
11
|
pubmed:dateCreated |
2003-10-27
|
pubmed:abstractText |
To examine the expression and regulation of chemotactic factor, macrophage inflammatory protein-1alpha (MIP-1alpha) by fibroblast-like synoviocytes (FLS), monocytes and polymorphonuclear neutrophils (PMN) isolated from the synovial fluid (SF) of rheumatoid arthritis (RA) patients.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Nov
|
pubmed:issn |
1462-0324
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
42
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1390-7
|
pubmed:dateRevised |
2007-11-15
|
pubmed:meshHeading |
pubmed-meshheading:12832710-Antigens, CD18,
pubmed-meshheading:12832710-Arthritis, Rheumatoid,
pubmed-meshheading:12832710-Blotting, Northern,
pubmed-meshheading:12832710-Cells, Cultured,
pubmed-meshheading:12832710-Chemokine CCL3,
pubmed-meshheading:12832710-Chemokine CCL4,
pubmed-meshheading:12832710-Enzyme-Linked Immunosorbent Assay,
pubmed-meshheading:12832710-Fibroblasts,
pubmed-meshheading:12832710-Humans,
pubmed-meshheading:12832710-Immunohistochemistry,
pubmed-meshheading:12832710-Intercellular Adhesion Molecule-1,
pubmed-meshheading:12832710-Lymphocyte Activation,
pubmed-meshheading:12832710-Macrophage Inflammatory Proteins,
pubmed-meshheading:12832710-Monocytes,
pubmed-meshheading:12832710-Neutrophils,
pubmed-meshheading:12832710-RNA, Messenger,
pubmed-meshheading:12832710-Synovial Membrane
|
pubmed:year |
2003
|
pubmed:articleTitle |
Activated leucocytes express and secrete macrophage inflammatory protein-1alpha upon interaction with synovial fibroblasts of rheumatoid arthritis via a beta2-integrin/ICAM-1 mechanism.
|
pubmed:affiliation |
Showa University School of Medicine, Tokyo, Japan.
|
pubmed:publicationType |
Journal Article,
Clinical Trial,
Controlled Clinical Trial,
Research Support, Non-U.S. Gov't
|