| pubmed-article:10894769 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0003864 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0040300 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C1533591 | lld:lifeskim |
| pubmed-article:10894769 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
| pubmed-article:10894769 | pubmed:issue | 5477 | lld:pubmed |
| pubmed-article:10894769 | pubmed:dateCreated | 2000-7-25 | lld:pubmed |
| pubmed-article:10894769 | pubmed:abstractText | Mutation at the mouse progressive ankylosis (ank) locus causes a generalized, progressive form of arthritis accompanied by mineral deposition, formation of bony outgrowths, and joint destruction. Here, we show that the ank locus encodes a multipass transmembrane protein (ANK) that is expressed in joints and other tissues and controls pyrophosphate levels in cultured cells. A highly conserved gene is present in humans and other vertebrates. These results identify ANK-mediated control of pyrophosphate levels as a possible mechanism regulating tissue calcification and susceptibility to arthritis in higher animals. | lld:pubmed |
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| pubmed-article:10894769 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10894769 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10894769 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10894769 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10894769 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:10894769 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:10894769 | pubmed:author | pubmed-author:JohnsonM DMD | lld:pubmed |
| pubmed-article:10894769 | pubmed:author | pubmed-author:KingsleyD MDM | lld:pubmed |
| pubmed-article:10894769 | pubmed:author | pubmed-author:HoA MAM | lld:pubmed |
| pubmed-article:10894769 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10894769 | pubmed:day | 14 | lld:pubmed |
| pubmed-article:10894769 | pubmed:volume | 289 | lld:pubmed |
| pubmed-article:10894769 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10894769 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10894769 | pubmed:pagination | 265-70 | lld:pubmed |
| pubmed-article:10894769 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:10894769 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:10894769 | pubmed:articleTitle | Role of the mouse ank gene in control of tissue calcification and arthritis. | lld:pubmed |
| pubmed-article:10894769 | pubmed:affiliation | Department of Developmental Biology and Howard Hughes Medical Institute, Beckman Center B300, Stanford University School of Medicine, Stanford, CA 94305-5327, USA. | lld:pubmed |
| pubmed-article:10894769 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10894769 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:10894769 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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