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| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0005821 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0039194 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0948089 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0002965 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0167627 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C1749467 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0699748 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C1314939 | lld:lifeskim |
| pubmed-article:10441098 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:10441098 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:10441098 | pubmed:dateCreated | 1999-9-13 | lld:pubmed |
| pubmed-article:10441098 | pubmed:abstractText | The CD40 ligand (CD40L) on activated T cells and platelets may be activating matrix metalloproteinases, inducing procoagulant activity, and be involved in the pathogenesis of acute coronary syndromes by promoting plaque rupture in atheroma. | lld:pubmed |
| pubmed-article:10441098 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:10441098 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:10441098 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10441098 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:10441098 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:MüllerFF | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:FrølandS SSS | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:ForfangKK | lld:pubmed |
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| pubmed-article:10441098 | pubmed:author | pubmed-author:GullestadLL | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:BergerVV | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:AukrustPP | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:BrunsvigAA | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:AaserEE | lld:pubmed |
| pubmed-article:10441098 | pubmed:author | pubmed-author:UelandTT | lld:pubmed |
| pubmed-article:10441098 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:10441098 | pubmed:day | 10 | lld:pubmed |
| pubmed-article:10441098 | pubmed:volume | 100 | lld:pubmed |
| pubmed-article:10441098 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10441098 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10441098 | pubmed:pagination | 614-20 | lld:pubmed |
| pubmed-article:10441098 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:10441098 | pubmed:year | 1999 | lld:pubmed |
| pubmed-article:10441098 | pubmed:articleTitle | Enhanced levels of soluble and membrane-bound CD40 ligand in patients with unstable angina. Possible reflection of T lymphocyte and platelet involvement in the pathogenesis of acute coronary syndromes. | lld:pubmed |
| pubmed-article:10441098 | pubmed:affiliation | Section of Clinical Immunology and Infectious Diseases, Research Institute for Internal Medicine, Department of Cardiology, University of Oslo, Norway. | lld:pubmed |
| pubmed-article:10441098 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:10441098 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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