Source:http://www.reactome.org/biopax/48887BiochemicalReaction368
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Authored: Gopinathrao, G, 2008-11-23 14:00:06,
Edited: Gopinathrao, G, 2008-11-24 14:15:13,
Regulation of epithelial chloride flux, which is defective in patients with cystic fibrosis, may be mediated by phosphorylation of the cystic fibrosis transmembrane conductance regulator (CFTR) by cyclic AMP-dependent protein kinase (PKA) or protein kinase C (PKC). CFTR regulates both HCO(3)(-) secretion and HCO(3)(-) salvage in secretory epithelia.,
Reviewed: Matthews, L, 2008-12-02 15:41:53
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HCO3- transport through ion channel
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