Source:http://linkedlifedata.com/resource/entrezgene/interaction/5295-155871
| Predicate | Object |
|---|---|
| rdf:type | |
| entrezgene:pubmed |
pubmed-article:11154208,
pubmed-article:11156964,
pubmed-article:11994280,
pubmed-article:12077252,
pubmed-article:14602571,
pubmed-article:17157319,
pubmed-article:20019835,
pubmed-article:21029719,
pubmed-article:21765914,
pubmed-article:8798481,
pubmed-article:9394803,
pubmed-article:9446795,
pubmed-article:9708406
|
| entrezgene:interactant | |
| entrezgene:geneRifText |
Activation of protein kinase C-epsilon by HIV-1 Tat involves an interaction with PI 3-kinase,
Arctigenin regulates the upstream PI3K enzyme from converting PIP2 to PIP3 in Tat-expressing CHME5 cells,
HIV-1 Tat activates phosphatidylinositol 3-kinase (PI3K), leading to the activation of Akt/PKB, decreased levels of cAMP and CREB phosphorylation, and the protection of cells from apoptosis,
HIV-1 Tat in combination with KSHV kaposin A activates the MEK/ERK, STAT3, and PI3K/Akt signals in NIH3T3 cells,
HIV-1 Tat induced release of MCP-1 from human lung microvascular endothelial cells involves an interaction between Tat and PI3 kinase,
HIV-1 Tat-mediated cytopathic changes in human brain micro vascular endothelial cells involves its interaction with phosphatidylinositol 3-kinase (PI3K),
Phosphatidylinositol 3-kinase inhibits HIV-1 Tat-mediated transactivation of the HIV-1 LTR promoter by affecting the formation of the Tat-associated kinase transactivating complex,
Phosphatidylinositol 3-kinase/Akt signaling pathway is involved in HIV-1 Tat-induced NADPH oxidase stimulation and LTR transactivation,
Pro-survival effects of intracellular HIV-1 Tat in a microglial cell line is attributed to activation of the PI-3-kinase (PI3K)/Akt pathway via decreasing expression of PTEN, a negative regulator of the PI-3-K pathway
|
| entrezgene:keyphrase |
interacts with,
inhibited by,
activates
|