Source:http://linkedlifedata.com/resource/entrezgene/hivinteraction/155971-3552
Predicate | Object |
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rdf:type | |
entrezgene:pubmed |
pubmed-article:12089333,
pubmed-article:15481145,
pubmed-article:1548758,
pubmed-article:16160188,
pubmed-article:16934308,
pubmed-article:1905933,
pubmed-article:1993354,
pubmed-article:2536171,
pubmed-article:7511078,
pubmed-article:7561697,
pubmed-article:7706435,
pubmed-article:7815507,
pubmed-article:8458986,
pubmed-article:8764000,
pubmed-article:8892361,
pubmed-article:8938574,
pubmed-article:9108403,
pubmed-article:9185130,
pubmed-article:9225992,
pubmed-article:9658081
|
entrezgene:interactant | |
entrezgene:geneRifText |
Addition of HIV-1 gp120 to IL-1 treated human granulocytes or monocytes results in a slower movement that is chemokinetic (loss of directionality or random) in nature,
Cytokines induced in vitro by HIV-1 gp120 in normal peripheral blood mononuclear cells (PBMC) include interferon-alpha (IFN-alpha) and IFN-gamma, tumor necrosis factor-alpha (TNF-alpha), IL-6, IL-10, IL-1 alpha and IL1 beta,
HIV-1 gp120 increases the levels of prostaglandin E2 (PGE2) and IL-1 in human cells,
HIV-1 gp41 or gp120 synthetic peptides induce the production of interleukin (IL)-1 and tumor necrosis factor (TNF); in contrast, gp41 or gp120 synthetic peptides are able to depress the production of interferon (IFN)-alpha, IFN-gamma, and IL-2,
IL-1 and TNF-alpha production are induced by the treatment of cells with HIV-1 gp160
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entrezgene:keyphrase |
interacts with,
upregulates
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