Source:http://linkedlifedata.com/resource/#_5033363133350019
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http://linkedlifedata.com/resource/#_503336313335001A,
http://linkedlifedata.com/resource/#_503336313335001B,
http://linkedlifedata.com/resource/#_503336313335001C,
http://linkedlifedata.com/resource/#_503336313335001D,
http://linkedlifedata.com/resource/#_503336313335001E,
http://linkedlifedata.com/resource/#_503336313335001F,
http://linkedlifedata.com/resource/#_5033363133350020
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| rdf:predicate | |
| rdf:object |
Involved in aging, oxidative stress response, and in the regulation of mitochondrial biogenesis. Inactivation of UTH1 increases life span, leads to higher resistance to heat stress and against hydrogen peroxide, and increases sensitivity to the superoxide radical-generating drug paraquat and to copper. Also required for the selective autophagic degradation of mitochondria (mitophagy) in response to nitrogen starvation. May play a role in cell wall morphogenesis and septation.
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