Statements in which the resource exists.
SubjectPredicateObjectContext
http://www.reactome.org/bio...rdf:typebiopax3:BiochemicalReactionlld:biopax3
http://www.reactome.org/bio...biopax3:commentEdited: Rothfels, K, 2012-05-16lld:biopax3
http://www.reactome.org/bio...biopax3:commentReviewed: Ezzat, S, 2012-05-15lld:biopax3
http://www.reactome.org/bio...biopax3:commentExpression of the FGFR4 Y367C mutant in MDA-MB453 breast cancer cell line results in constitutive tyrosine phosphorylation of the receptor and serum-independent activation of downstream signaling as monitored by Erk phosphorylation. Ectopic expression of FGFR4 Y367C in HEK cells also leads to Erk activation and enhanced cellular proliferation. Akt and phospho-AKT levels were not affected by overexpression of the FGFR4 Y367C mutant, however (Roidl, 2010)lld:biopax3
http://www.reactome.org/bio...biopax3:commentAuthored: Rothfels, K, 2012-02-09lld:biopax3
http://www.reactome.org/bio...biopax3:xrefhttp://identifiers.org/pubm...lld:biopax3
http://www.reactome.org/bio...biopax3:xrefurn:biopax:UnificationXref:...lld:biopax3
http://www.reactome.org/bio...biopax3:xrefurn:biopax:UnificationXref:...lld:biopax3
http://www.reactome.org/bio...biopax3:dataSourceurn:biopax:Provenance:react...lld:biopax3
http://www.reactome.org/bio...biopax3:dataSourcehttp://www.reactome.org/bio...lld:biopax3
http://www.reactome.org/bio...biopax3:displayNameAutocatalytic phosphorylation of FGFR4 Y367C mutantlld:biopax3
http://www.reactome.org/bio...biopax3:eCNumber2.7.10lld:biopax3
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http://www.reactome.org/bio...biopax3:pathwayComponenthttp://www.reactome.org/bio...lld:biopax3